CHD1L prevents lipopolysaccharide-induced hepatocellular carcinomar cell death by activating hnRNP A2/B1-nmMYLK axis.

Wang, Guangliang; Zhang, Xiaofeng; Cheng, Wei; Mo, Yanxuan; Chen, Juan; Cao, Zhiming; Chen, Xiaogang; Cui, Huiqin; Liu, Shanshan; Huang, Li; Liu, Ming; Ma, Lei; Ma, Ning-Fang

Wang, Guangliang; Zhang, Xiaofeng; Cheng, Wei; Mo, Yanxuan; Chen, Juan; Cao, Zhiming; Chen, Xiaogang; Cui, Huiqin; Liu, Shanshan; Huang, Li; Liu, Ming; Ma, Lei; Ma, Ning-Fang.

Afiliação

Wang G; Affiliated Cancer Hospital and Institute, Guangzhou Medical University, Guangzhou, China.
Zhang X; Department of Histology and Embryology, Faculty of Basic Medical Sciences, Guilin Medical University, Guilin, Guangxi, China.
Cheng W; Affiliated Cancer Hospital and Institute, Guangzhou Medical University, Guangzhou, China.
Mo Y; Guangzhou Municipal and Guangdong Provincial Key Laboratory of Protein Modification and Degradation, School of Basic Medical Sciences, Guangzhou Medical University, Guangzhou, China.
Chen J; Guangzhou Municipal and Guangdong Provincial Key Laboratory of Protein Modification and Degradation, School of Basic Medical Sciences, Guangzhou Medical University, Guangzhou, China.
Cao Z; Guangzhou Municipal and Guangdong Provincial Key Laboratory of Protein Modification and Degradation, School of Basic Medical Sciences, Guangzhou Medical University, Guangzhou, China.
Chen X; Affiliated Cancer Hospital and Institute, Guangzhou Medical University, Guangzhou, China.
Cui H; Affiliated Cancer Hospital and Institute, Guangzhou Medical University, Guangzhou, China.
Liu S; Guangzhou Municipal and Guangdong Provincial Key Laboratory of Protein Modification and Degradation, School of Basic Medical Sciences, Guangzhou Medical University, Guangzhou, China.
Huang L; Affiliated Cancer Hospital and Institute, Guangzhou Medical University, Guangzhou, China.
Liu M; Guangzhou Municipal and Guangdong Provincial Key Laboratory of Protein Modification and Degradation, School of Basic Medical Sciences, Guangzhou Medical University, Guangzhou, China.
Ma L; Guangzhou Municipal and Guangdong Provincial Key Laboratory of Protein Modification and Degradation, School of Basic Medical Sciences, Guangzhou Medical University, Guangzhou, China.
Ma NF; Guangzhou Municipal and Guangdong Provincial Key Laboratory of Protein Modification and Degradation, School of Basic Medical Sciences, Guangzhou Medical University, Guangzhou, China.

Cell Death Dis ; 12(10): 891, 2021 09 29.

Article em En | MEDLINE | ID: mdl-34588420

Assuntos

Proteínas de Ligação ao Cálcio/metabolismo; Carcinoma Hepatocelular/patologia; DNA Helicases/metabolismo; Proteínas de Ligação a DNA/metabolismo; Ribonucleoproteínas Nucleares Heterogêneas Grupo A-B/metabolismo; Neoplasias Hepáticas/patologia; Quinase de Cadeia Leve de Miosina/metabolismo; Animais; Apoptose/genética; Sequência de Bases; Carcinoma Hepatocelular/genética; Morte Celular; Proliferação de Células/genética; DNA Helicases/genética; Proteínas de Ligação a DNA/genética; Regulação para Baixo/genética; Feminino; Regulação Neoplásica da Expressão Gênica; Células Hep G2; Humanos; Lipopolissacarídeos; Neoplasias Hepáticas/genética; Masculino; Camundongos Endogâmicos BALB C; Camundongos Nus; Pessoa de Meia-Idade; Modelos Biológicos; NF-kappa B/metabolismo; Transdução de Sinais; Receptor 4 Toll-Like/metabolismo

Texto completo

Imprimir

XML

PubMed Links

Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Quinase de Cadeia Leve de Miosina / Proteínas de Ligação ao Cálcio / DNA Helicases / Carcinoma Hepatocelular / Ribonucleoproteínas Nucleares Heterogêneas Grupo A-B / Proteínas de Ligação a DNA / Neoplasias Hepáticas Tipo de estudo: Prognostic_studies Limite: Animals / Female / Humans / Male / Middle aged Idioma: En Ano de publicação: 2021 Tipo de documento: Article

Texto completo

Imprimir

XML

PubMed Links

Buscar no Google