GB83, an Agonist of PAR2 with a Unique Mechanism of Action Distinct from Trypsin and PAR2-AP.

Heo, Yunkyung; Yang, Eunhee; Lee, Yechan; Seo, Yohan; Ryu, Kunhi; Jeon, Hyejin; Namkung, Wan

Heo, Yunkyung; Yang, Eunhee; Lee, Yechan; Seo, Yohan; Ryu, Kunhi; Jeon, Hyejin; Namkung, Wan.

Afiliação

Heo Y; College of Pharmacy, Yonsei Institute of Pharmaceutical Sciences, Yonsei University, Incheon 21983, Korea.
Yang E; College of Pharmacy, Yonsei Institute of Pharmaceutical Sciences, Yonsei University, Incheon 21983, Korea.
Lee Y; College of Pharmacy, Yonsei Institute of Pharmaceutical Sciences, Yonsei University, Incheon 21983, Korea.
Seo Y; College of Pharmacy, Yonsei Institute of Pharmaceutical Sciences, Yonsei University, Incheon 21983, Korea.
Ryu K; New Drug Development Center, Daegu Gyeongbuk Medical Innovation Foundation, Daegu 41061, Korea.
Jeon H; College of Pharmacy, Yonsei Institute of Pharmaceutical Sciences, Yonsei University, Incheon 21983, Korea.
Namkung W; College of Pharmacy, Yonsei Institute of Pharmaceutical Sciences, Yonsei University, Incheon 21983, Korea.

Int J Mol Sci ; 23(18)2022 Sep 13.

Article em En | MEDLINE | ID: mdl-36142527

Assuntos

Cálcio; Receptor PAR-2; Cálcio/metabolismo; Peptídeos; Receptor PAR-2/metabolismo; Tripsina; beta-Arrestinas

Palavras-chave

GB83; PAR2; agonist; desensitization

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Cálcio / Receptor PAR-2 Tipo de estudo: Prognostic_studies Idioma: En Ano de publicação: 2022 Tipo de documento: Article

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