Mitochondria dysfunction and impaired response to oxidative stress promotes proteostasis disruption in aged human cells.

Trigo, Diogo; Nadais, André; Carvalho, Ana; Morgado, Bárbara; Santos, Francisco; Nóbrega-Pereira, Sandrina; da Cruz E Silva, Odete A B

Trigo, Diogo; Nadais, André; Carvalho, Ana; Morgado, Bárbara; Santos, Francisco; Nóbrega-Pereira, Sandrina; da Cruz E Silva, Odete A B.

Afiliação

Trigo D; Institute of Biomedicine (iBiMED), Department of Medical Sciences, University of Aveiro, 3810-193 Aveiro, Portugal. Electronic address: trigo.diogo@ua.pt.
Nadais A; Institute of Biomedicine (iBiMED), Department of Medical Sciences, University of Aveiro, 3810-193 Aveiro, Portugal.
Carvalho A; Institute of Biomedicine (iBiMED), Department of Medical Sciences, University of Aveiro, 3810-193 Aveiro, Portugal.
Morgado B; Institute of Biomedicine (iBiMED), Department of Medical Sciences, University of Aveiro, 3810-193 Aveiro, Portugal.
Santos F; Institute of Biomedicine (iBiMED), Department of Medical Sciences, University of Aveiro, 3810-193 Aveiro, Portugal.
Nóbrega-Pereira S; Institute of Biomedicine (iBiMED), Department of Medical Sciences, University of Aveiro, 3810-193 Aveiro, Portugal.
da Cruz E Silva OAB; Institute of Biomedicine (iBiMED), Department of Medical Sciences, University of Aveiro, 3810-193 Aveiro, Portugal.

Mitochondrion ; 69: 1-9, 2023 03.

Article em En | MEDLINE | ID: mdl-36273801

Assuntos

Agregados Proteicos; Proteostase; Humanos; Idoso; Estresse Oxidativo; Mitocôndrias/metabolismo; Envelhecimento/metabolismo; Antioxidantes/metabolismo

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Agregados Proteicos / Proteostase Limite: Aged / Humans Idioma: En Ano de publicação: 2023 Tipo de documento: Article

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