Could senescence phenotypes strike the balance to promote tumor dormancy?

Chiu, Fang-Yen; Kvadas, Raegan M; Mheidly, Zeinab; Shahbandi, Ashkan; Jackson, James G

Chiu, Fang-Yen; Kvadas, Raegan M; Mheidly, Zeinab; Shahbandi, Ashkan; Jackson, James G.

Afiliação

Chiu FY; Department of Biochemistry and Molecular Biology, Tulane School of Medicine, 1430 Tulane Avenue, New Orleans, LA, 70112, USA.
Kvadas RM; Department of Biochemistry and Molecular Biology, Tulane School of Medicine, 1430 Tulane Avenue, New Orleans, LA, 70112, USA.
Mheidly Z; Department of Biochemistry and Molecular Biology, Tulane School of Medicine, 1430 Tulane Avenue, New Orleans, LA, 70112, USA.
Shahbandi A; Department of Biochemistry and Molecular Biology, Tulane School of Medicine, 1430 Tulane Avenue, New Orleans, LA, 70112, USA.
Jackson JG; Department of Biochemistry and Molecular Biology, Tulane School of Medicine, 1430 Tulane Avenue, New Orleans, LA, 70112, USA. jjacks8@tulane.edu.

Cancer Metastasis Rev ; 42(1): 143-160, 2023 03.

Article em En | MEDLINE | ID: mdl-36735097

Assuntos

Recidiva Local de Neoplasia; Neoplasias; Humanos; Recidiva Local de Neoplasia/patologia; Neoplasias/metabolismo; Fenótipo; Senescência Celular; Citocinas/uso terapêutico; Microambiente Tumoral

Palavras-chave

Angiogenesis; Breast cancer; Cellular senescence; Chromatin; Immune evasion; Tumor dormancy; p53

Texto completo

Imprimir

XML

PubMed Links

Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Recidiva Local de Neoplasia / Neoplasias Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Ano de publicação: 2023 Tipo de documento: Article

Texto completo

Imprimir

XML

PubMed Links

Buscar no Google