Inflammation differentially controls transport of depolarizing Nav versus hyperpolarizing Kv channels to drive rat nociceptor activity.

Higerd-Rusli, Grant P; Tyagi, Sidharth; Baker, Christopher A; Liu, Shujun; Dib-Hajj, Fadia B; Dib-Hajj, Sulayman D; Waxman, Stephen G

Higerd-Rusli, Grant P; Tyagi, Sidharth; Baker, Christopher A; Liu, Shujun; Dib-Hajj, Fadia B; Dib-Hajj, Sulayman D; Waxman, Stephen G.

Afiliação

Higerd-Rusli GP; Medical Scientist Training Program, Yale University School of Medicine, New Haven, CT 06520.
Tyagi S; Center for Neuroscience and Regeneration Research, Yale University School of Medicine, New Haven, CT 06510.
Baker CA; Department of Neurology, Yale University School of Medicine, New Haven, CT 06510.
Liu S; Rehabilitation Research Center, Veterans Affairs Connecticut Healthcare System, West Haven, CT 06516.
Dib-Hajj FB; Cellular and Molecular Physiology Graduate Program, Yale University School of Medicine, New Haven, CT 06520.
Dib-Hajj SD; Medical Scientist Training Program, Yale University School of Medicine, New Haven, CT 06520.
Waxman SG; Center for Neuroscience and Regeneration Research, Yale University School of Medicine, New Haven, CT 06510.

Proc Natl Acad Sci U S A ; 120(11): e2215417120, 2023 03 14.

Article em En | MEDLINE | ID: mdl-36897973

Assuntos

Axônios; Nociceptores; Ratos; Animais; Axônios/metabolismo; Dor/metabolismo; Inflamação/metabolismo; Mediadores da Inflamação/metabolismo

Palavras-chave

inflammation; intracellular trafficking; ion channels; neuronal excitability; pain

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Axônios / Nociceptores Limite: Animals Idioma: En Ano de publicação: 2023 Tipo de documento: Article

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