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Presynaptic targeting of botulinum neurotoxin type A requires a tripartite PSG-Syt1-SV2 plasma membrane nanocluster for synaptic vesicle entry.
Joensuu, Merja; Syed, Parnayan; Saber, Saber H; Lanoue, Vanessa; Wallis, Tristan P; Rae, James; Blum, Ailisa; Gormal, Rachel S; Small, Christopher; Sanders, Shanley; Jiang, Anmin; Mahrhold, Stefan; Krez, Nadja; Cousin, Michael A; Cooper-White, Ruby; Cooper-White, Justin J; Collins, Brett M; Parton, Robert G; Balistreri, Giuseppe; Rummel, Andreas; Meunier, Frédéric A.
Afiliação
  • Joensuu M; Clem Jones Centre for Ageing Dementia Research, Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Syed P; Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Saber SH; Australian Institute for Bioengineering and Nanotechnology, The University of Queensland, Brisbane, QLD, Australia.
  • Lanoue V; Clem Jones Centre for Ageing Dementia Research, Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Wallis TP; Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Rae J; Clem Jones Centre for Ageing Dementia Research, Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Blum A; Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Gormal RS; Australian Institute for Bioengineering and Nanotechnology, The University of Queensland, Brisbane, QLD, Australia.
  • Small C; Clem Jones Centre for Ageing Dementia Research, Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Sanders S; Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Jiang A; Clem Jones Centre for Ageing Dementia Research, Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Mahrhold S; Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Krez N; Institute for Molecular Bioscience, The University of Queensland, Brisbane, QLD, Australia.
  • Cousin MA; Clem Jones Centre for Ageing Dementia Research, Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Cooper-White R; Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Cooper-White JJ; Clem Jones Centre for Ageing Dementia Research, Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Collins BM; Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Parton RG; Clem Jones Centre for Ageing Dementia Research, Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Balistreri G; Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Rummel A; Clem Jones Centre for Ageing Dementia Research, Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
  • Meunier FA; Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.
EMBO J ; 42(13): e112095, 2023 07 03.
Article em En | MEDLINE | ID: mdl-37226896
The unique nerve terminal targeting of botulinum neurotoxin type A (BoNT/A) is due to its capacity to bind two receptors on the neuronal plasma membrane: polysialoganglioside (PSG) and synaptic vesicle glycoprotein 2 (SV2). Whether and how PSGs and SV2 may coordinate other proteins for BoNT/A recruitment and internalization remains unknown. Here, we demonstrate that the targeted endocytosis of BoNT/A into synaptic vesicles (SVs) requires a tripartite surface nanocluster. Live-cell super-resolution imaging and electron microscopy of catalytically inactivated BoNT/A wildtype and receptor-binding-deficient mutants in cultured hippocampal neurons demonstrated that BoNT/A must bind coincidentally to a PSG and SV2 to target synaptic vesicles. We reveal that BoNT/A simultaneously interacts with a preassembled PSG-synaptotagmin-1 (Syt1) complex and SV2 on the neuronal plasma membrane, facilitating Syt1-SV2 nanoclustering that controls endocytic sorting of the toxin into synaptic vesicles. Syt1 CRISPRi knockdown suppressed BoNT/A- and BoNT/E-induced neurointoxication as quantified by SNAP-25 cleavage, suggesting that this tripartite nanocluster may be a unifying entry point for selected botulinum neurotoxins that hijack this for synaptic vesicle targeting.
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Toxinas Botulínicas Tipo A Limite: Animals Idioma: En Ano de publicação: 2023 Tipo de documento: Article

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Toxinas Botulínicas Tipo A Limite: Animals Idioma: En Ano de publicação: 2023 Tipo de documento: Article