LTK and ALK promote neuronal polarity and cortical migration by inhibiting IGF1R activity.

Christova, Tania; Ho, Stephanie Ky; Liu, Ying; Gill, Mandeep; Attisano, Liliana

Christova, Tania; Ho, Stephanie Ky; Liu, Ying; Gill, Mandeep; Attisano, Liliana.

Afiliação

Christova T; Department of Biochemistry, Donnelly Centre, University of Toronto, Toronto, ON, Canada.
Ho SK; Department of Biochemistry, Donnelly Centre, University of Toronto, Toronto, ON, Canada.
Liu Y; Department of Biochemistry, Donnelly Centre, University of Toronto, Toronto, ON, Canada.
Gill M; Department of Biochemistry, Donnelly Centre, University of Toronto, Toronto, ON, Canada.
Attisano L; Department of Biochemistry, Donnelly Centre, University of Toronto, Toronto, ON, Canada.

EMBO Rep ; 24(7): e56937, 2023 Jul 05.

Article em En | MEDLINE | ID: mdl-37291945

Assuntos

Neurônios; Receptores Proteína Tirosina Quinases; Animais; Camundongos; Axônios/metabolismo; Polaridade Celular; Neurogênese/genética; Neurônios/metabolismo; Receptores Proteína Tirosina Quinases/genética; Receptores Proteína Tirosina Quinases/metabolismo; Transdução de Sinais

Palavras-chave

ALK; IGF-1R; LTK; cortical migration; neuronal polarity

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Receptores Proteína Tirosina Quinases / Neurônios Limite: Animals Idioma: En Ano de publicação: 2023 Tipo de documento: Article

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