Your browser doesn't support javascript.
loading
Keap1 loss promotes Kras-driven lung cancer and results in dependence on glutaminolysis.
Romero, Rodrigo; Sayin, Volkan I; Davidson, Shawn M; Bauer, Matthew R; Singh, Simranjit X; LeBoeuf, Sarah E; Karakousi, Triantafyllia R; Ellis, Donald C; Bhutkar, Arjun; Sánchez-Rivera, Francisco J; Subbaraj, Lakshmipriya; Martinez, Britney; Bronson, Roderick T; Prigge, Justin R; Schmidt, Edward E; Thomas, Craig J; Goparaju, Chandra; Davies, Angela; Dolgalev, Igor; Heguy, Adriana; Allaj, Viola; Poirier, John T; Moreira, Andre L; Rudin, Charles M; Pass, Harvey I; Vander Heiden, Matthew G; Jacks, Tyler; Papagiannakopoulos, Thales.
Afiliação
  • Romero R; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
  • Sayin VI; Department of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
  • Davidson SM; Department of Pathology, New York University School of Medicine, New York, New York, USA.
  • Bauer MR; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
  • Singh SX; Department of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
  • LeBoeuf SE; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
  • Karakousi TR; Department of Pathology, New York University School of Medicine, New York, New York, USA.
  • Ellis DC; Department of Pathology, New York University School of Medicine, New York, New York, USA.
  • Bhutkar A; Department of Pathology, New York University School of Medicine, New York, New York, USA.
  • Sánchez-Rivera FJ; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
  • Subbaraj L; Department of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
  • Martinez B; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
  • Bronson RT; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
  • Prigge JR; Department of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
  • Schmidt EE; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
  • Thomas CJ; Department of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
  • Goparaju C; Department of Pathology, New York University School of Medicine, New York, New York, USA.
  • Davies A; Tufts University, Boston, Massachusetts, USA.
  • Dolgalev I; Harvard Medical School, Boston, Massachusetts, USA.
  • Heguy A; Department of Immunology and Infectious Diseases, Montana State University, Bozeman, Montana, USA.
  • Allaj V; Department of Immunology and Infectious Diseases, Montana State University, Bozeman, Montana, USA.
  • Poirier JT; National Institutes of Health Chemical Genomics Center, Division of Preclinical Innovation, National Center for Advancing Translational Sciences, US National Institutes of Health, Bethesda, Maryland, USA.
  • Moreira AL; Department of Cardiothoracic Surgery, New York University Langone Medical Center, New York, New York, USA.
  • Rudin CM; Champions Oncology, Hackensack, New Jersey, USA.
  • Pass HI; Genome Technology Center, New York University School of Medicine, New York, New York, USA.
  • Vander Heiden MG; Genome Technology Center, New York University School of Medicine, New York, New York, USA.
  • Jacks T; Molecular Pharmacology Program, Memorial Sloan Kettering Cancer Center, New York, New York, USA.
  • Papagiannakopoulos T; Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, New York, USA.
Nat Med ; 23(11): 1362-1368, 2017 Nov.
Article em En | MEDLINE | ID: mdl-28967920
Assuntos
Texto completo
Adicionar na Minha BVS
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Adenocarcinoma / Genes ras / Proteína 1 Associada a ECH Semelhante a Kelch / Glutamina / Neoplasias Pulmonares Tipo de estudo: Prognostic_studies Limite: Animals / Humans Idioma: En Revista: Nat Med Assunto da revista: BIOLOGIA MOLECULAR / MEDICINA Ano de publicação: 2017 Tipo de documento: Article País de afiliação: Estados Unidos País de publicação: Estados Unidos
Texto completo
Adicionar na Minha BVS
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Adenocarcinoma / Genes ras / Proteína 1 Associada a ECH Semelhante a Kelch / Glutamina / Neoplasias Pulmonares Tipo de estudo: Prognostic_studies Limite: Animals / Humans Idioma: En Revista: Nat Med Assunto da revista: BIOLOGIA MOLECULAR / MEDICINA Ano de publicação: 2017 Tipo de documento: Article País de afiliação: Estados Unidos País de publicação: Estados Unidos