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1.
Bioorg Med Chem Lett ; 19(16): 4777-80, 2009 Aug 15.
Artigo em Inglês | MEDLINE | ID: mdl-19595590

RESUMO

Using the X-ray crystal structure of an amide-based progesterone receptor (PR) partial agonist bound to the PR ligand binding domain, a novel PR partial agonist class containing a pyrrolidine ring was designed. Members of this class of N-alkylpyrrolidines demonstrate potent and highly selective partial agonism of the progesterone receptor, and one of these analogs was shown to be efficacious upon oral dosing in the OVX rat model of estrogen opposition.


Assuntos
Pirrolidinas/química , Receptores de Progesterona/agonistas , Administração Oral , Animais , Sítios de Ligação , Simulação por Computador , Cristalografia por Raios X , Desenho de Fármacos , Modelos Animais , Estrutura Terciária de Proteína , Pirrolidinas/administração & dosagem , Pirrolidinas/síntese química , Ratos , Receptores de Progesterona/metabolismo
2.
Bioorg Med Chem Lett ; 19(10): 2637-41, 2009 May 15.
Artigo em Inglês | MEDLINE | ID: mdl-19376703

RESUMO

Two classes of amino acid-derived heterocyclic progesterone receptor ligands were developed to address the metabolic issues posed by the dimethyl amide functionality of the lead compound (1). The tetrazole-derived ligands behaved as potent partial agonists, while the 1,2,4-triazole ligands behaved as potent full agonists.


Assuntos
Receptores de Progesterona/agonistas , Tetrazóis/síntese química , Aminoácidos/química , Animais , Ratos , Receptores de Progesterona/metabolismo , Relação Estrutura-Atividade , Tetrazóis/química , Tetrazóis/farmacocinética
3.
Bioorg Med Chem Lett ; 19(16): 4664-8, 2009 Aug 15.
Artigo em Inglês | MEDLINE | ID: mdl-19616429

RESUMO

We have designed and synthesized a novel series of pyrrolidinones as progesterone receptor partial agonists. Compounds from this series had improved AR selectivity, rat pharmacokinetic properties, and in vivo potency compared to the lead compound. In addition, these compounds had improved selectivity against hERG channel inhibition.


Assuntos
Pirrolidinonas/química , Receptores de Progesterona/agonistas , Administração Oral , Animais , Sítios de Ligação , Descoberta de Drogas , Canais de Potássio Éter-A-Go-Go/metabolismo , Haplorrinos , Humanos , Pirrolidinonas/síntese química , Pirrolidinonas/farmacocinética , Ratos , Receptores de Progesterona/metabolismo , Relação Estrutura-Atividade
4.
Steroids ; 74(13-14): 1015-24, 2009.
Artigo em Inglês | MEDLINE | ID: mdl-19665469

RESUMO

Endometriosis, defined as the presence of endometrial glands and stroma at extra-uterine sites, is a gynecological condition that affects women of reproductive age. Consistent with its uterine origins, endometriotic lesions and resulting symptoms are hormonally responsive. To investigate Progesterone Receptor (PR)-based therapies, we measured physiological endpoints and gene expression in rat models of uterine cell estrogenic activity. Estrogen-induced ELT-3 rat leiomyoma cell proliferation was significantly inhibited by progesterone (P4), while the antiprogestin RU486 or the Selective PR Modulator (SPRM) asoprisnil, did not block proliferation. Stromal cell-derived factor-1 (SDF-1/Cxcl12) gene expression was induced by estrogen, and was repressed by the Selective Estrogen Receptor Modulators (SERMs), the antiestrogen ICI 182,780, and P4, but not by RU486 or the ERbeta-selective ligand ERB-041. In ELT-3 cells, asoprisnil demonstrated partial PR agonism on SDF-1 gene repression. Magnetic Resonance Imaging was used to monitor development of ectopic cysts in a rat surgical model of endometriosis. SERMs and P4 significantly decreased cyst volumes comparably by approximately 60%. However, ERB-041 and asoprisnil had no effect on cyst volume, and RU486 increased cyst volume by 20%. SDF-1 expression was modestly, but significantly, increased in the cyst compared to eutopic uterus, and P4 and raloxifene could repress the expression. We showed that SDF-1 was similarly regulated in human cells. These data suggest that transcriptional regulation of SDF-1 is a surrogate marker of estrogenic activities via ERalpha in rat uterine cells, and that SDF-1 repression by PR agonists can predict the ability to oppose the actions of estrogen in vivo.


Assuntos
Quimiocina CXCL12/antagonistas & inibidores , Endometriose/tratamento farmacológico , Progesterona/uso terapêutico , Progestinas/uso terapêutico , Receptores de Progesterona/agonistas , Moduladores Seletivos de Receptor Estrogênico/uso terapêutico , Útero/efeitos dos fármacos , Animais , Linhagem Celular Tumoral , Proliferação de Células/efeitos dos fármacos , Quimiocina CXCL12/agonistas , Quimiocina CXCL12/metabolismo , Cistos/tratamento farmacológico , Cistos/metabolismo , Cistos/patologia , Modelos Animais de Doenças , Endometriose/metabolismo , Endometriose/patologia , Estradiol/análogos & derivados , Estradiol/farmacologia , Estrenos/farmacologia , Antagonistas de Estrogênios/farmacologia , Receptor alfa de Estrogênio/metabolismo , Estrogênios/farmacologia , Feminino , Fulvestranto , Expressão Gênica , Antagonistas de Hormônios/farmacologia , Humanos , Mifepristona/farmacologia , Oxazóis/farmacologia , Oximas/farmacologia , Progesterona/farmacologia , Progestinas/farmacologia , Ratos , Moduladores Seletivos de Receptor Estrogênico/farmacologia , Útero/metabolismo
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