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1.
Control of morphogenesis, protease secretion and gene expression in Candida albicans by the preferred nitrogen source ammonium.
Microbiology (Reading)
; 160(Pt 8): 1599-1608, 2014 Aug.
Article
in English
| MEDLINE | ID: mdl-24841705
2.
Roles of different peptide transporters in nutrient acquisition in Candida albicans.
Eukaryot Cell
; 12(4): 520-8, 2013 Apr.
Article
in English
| MEDLINE | ID: mdl-23376942
3.
The stepwise acquisition of fluconazole resistance mutations causes a gradual loss of fitness in Candida albicans.
Mol Microbiol
; 86(3): 539-56, 2012 Nov.
Article
in English
| MEDLINE | ID: mdl-22924823
4.
Loss of heterozygosity at an unlinked genomic locus is responsible for the phenotype of a Candida albicans sap4Δ sap5Δ sap6Δ mutant.
Eukaryot Cell
; 10(1): 54-62, 2011 Jan.
Article
in English
| MEDLINE | ID: mdl-21097666
5.
Role of the Npr1 kinase in ammonium transport and signaling by the ammonium permease Mep2 in Candida albicans.
Eukaryot Cell
; 10(3): 332-42, 2011 Mar.
Article
in English
| MEDLINE | ID: mdl-21278231
6.
Regulation of efflux pump expression and drug resistance by the transcription factors Mrr1, Upc2, and Cap1 in Candida albicans.
Antimicrob Agents Chemother
; 55(5): 2212-23, 2011 May.
Article
in English
| MEDLINE | ID: mdl-21402859
7.
Mutations in the multi-drug resistance regulator MRR1, followed by loss of heterozygosity, are the main cause of MDR1 overexpression in fluconazole-resistant Candida albicans strains.
Mol Microbiol
; 69(4): 827-40, 2008 Aug.
Article
in English
| MEDLINE | ID: mdl-18577180
8.
A gain-of-function mutation in the transcription factor Upc2p causes upregulation of ergosterol biosynthesis genes and increased fluconazole resistance in a clinical Candida albicans isolate.
Eukaryot Cell
; 7(7): 1180-90, 2008 Jul.
Article
in English
| MEDLINE | ID: mdl-18487346
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