Search details
1.
Impaired distal renal potassium handling in streptozotocin-induced diabetic mice.
Am J Physiol Renal Physiol
; 2024 May 23.
Article
in English
| MEDLINE | ID: mdl-38779755
2.
Deletion of renal Nedd4-2 abolishes the effect of high sodium intake (HS) on Kir4.1, ENaC, and NCC and causes hypokalemia during high HS.
Am J Physiol Renal Physiol
; 320(5): F883-F896, 2021 05 01.
Article
in English
| MEDLINE | ID: mdl-33818128
3.
Deletion of Kir5.1 abolishes the effect of high Na+ intake on Kir4.1 and Na+-Cl- cotransporter.
Am J Physiol Renal Physiol
; 320(6): F1045-F1058, 2021 06 01.
Article
in English
| MEDLINE | ID: mdl-33900854
4.
Renal Tubule Nedd4-2 Deficiency Stimulates Kir4.1/Kir5.1 and Thiazide-Sensitive NaCl Cotransporter in Distal Convoluted Tubule.
J Am Soc Nephrol
; 31(6): 1226-1242, 2020 06.
Article
in English
| MEDLINE | ID: mdl-32295826
5.
Kir4.1/Kir5.1 Activity Is Essential for Dietary Sodium Intake-Induced Modulation of Na-Cl Cotransporter.
J Am Soc Nephrol
; 30(2): 216-227, 2019 02.
Article
in English
| MEDLINE | ID: mdl-30559144
6.
Deletion of Kir5.1 Impairs Renal Ability to Excrete Potassium during Increased Dietary Potassium Intake.
J Am Soc Nephrol
; 30(8): 1425-1438, 2019 08.
Article
in English
| MEDLINE | ID: mdl-31239388
7.
Kir5.1 regulates Nedd4-2-mediated ubiquitination of Kir4.1 in distal nephron.
Am J Physiol Renal Physiol
; 315(4): F986-F996, 2018 10 01.
Article
in English
| MEDLINE | ID: mdl-29897283
8.
Role of WNK4 and kidney-specific WNK1 in mediating the effect of high dietary K+ intake on ROMK channel in the distal convoluted tubule.
Am J Physiol Renal Physiol
; 315(2): F223-F230, 2018 08 01.
Article
in English
| MEDLINE | ID: mdl-29667910
9.
Potassium intake modulates the thiazide-sensitive sodium-chloride cotransporter (NCC) activity via the Kir4.1 potassium channel.
Kidney Int
; 93(4): 893-902, 2018 04.
Article
in English
| MEDLINE | ID: mdl-29310825
10.
Nitric oxide inhibits basolateral 10-pS Cl- channels through the cGMP/PKG signaling pathway in the thick ascending limb of C57BL/6 mice.
Am J Physiol Renal Physiol
; 310(8): F755-F762, 2016 04 15.
Article
in English
| MEDLINE | ID: mdl-26764200
11.
Prognostic significance of VEGF-C, semaphorin 3F, and neuropilin-2 expression in oral squamous cell carcinomas and their relationship with lymphangiogenesis.
J Surg Oncol
; 111(4): 382-8, 2015 Mar 15.
Article
in English
| MEDLINE | ID: mdl-25475162
12.
Ferroptosis: a potential bridge linking gut microbiota and chronic kidney disease.
Cell Death Discov
; 10(1): 234, 2024 May 15.
Article
in English
| MEDLINE | ID: mdl-38750055
13.
Emerging role of antidiabetic drugs in cardiorenal protection.
Front Pharmacol
; 15: 1349069, 2024.
Article
in English
| MEDLINE | ID: mdl-38384297
14.
Defective natriuresis contributes to hyperkalemia in db/db mice during potassium supplementation.
J Hypertens
; 2024 May 21.
Article
in English
| MEDLINE | ID: mdl-38780161
15.
Expression of αvß6 integrin and collagen fibre in oral squamous cell carcinoma: association with clinical outcomes and prognostic implications.
J Oral Pathol Med
; 42(7): 547-56, 2013 Aug.
Article
in English
| MEDLINE | ID: mdl-23331428
16.
Inhibitory effect of S-nitroso-N-acetylpenicillamine on the basolateral 10-pS Cl- channel in thick ascending limb.
PLoS One
; 18(4): e0284707, 2023.
Article
in English
| MEDLINE | ID: mdl-37083928
17.
Gut microbiota and its metabolites - molecular mechanisms and management strategies in diabetic kidney disease.
Front Immunol
; 14: 1124704, 2023.
Article
in English
| MEDLINE | ID: mdl-36742307
18.
Activation of Kir4.1/Kir5.1 contributes to the cyclosporin A-induced stimulation of the renal NaCl cotransporter and hyperkalemic hypertension.
Acta Physiol (Oxf)
; 238(2): e13948, 2023 06.
Article
in English
| MEDLINE | ID: mdl-36764674
19.
Kir4.1 deletion prevents salt-sensitive hypertension in early streptozotocin-induced diabetic mice via Na + -Cl - cotransporter in the distal convoluted tubule.
J Hypertens
; 41(6): 958-970, 2023 06 01.
Article
in English
| MEDLINE | ID: mdl-37016934
20.
Single-cell transcriptomics: A new tool for studying diabetic kidney disease.
Front Physiol
; 13: 1053850, 2022.
Article
in English
| MEDLINE | ID: mdl-36685214