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1.
Hematopoietic-Derived Galectin-3 Causes Cellular and Systemic Insulin Resistance.
Cell
; 167(4): 973-984.e12, 2016 11 03.
Article
in English
| MEDLINE | ID: mdl-27814523
2.
A zebrafish embryo culture system defines factors that promote vertebrate myogenesis across species.
Cell
; 155(4): 909-921, 2013 Nov 07.
Article
in English
| MEDLINE | ID: mdl-24209627
3.
Myotubes derived from human-induced pluripotent stem cells mirror in vivo insulin resistance.
Proc Natl Acad Sci U S A
; 113(7): 1889-94, 2016 Feb 16.
Article
in English
| MEDLINE | ID: mdl-26831110
4.
A Cell-Autonomous Signature of Dysregulated Protein Phosphorylation Underlies Muscle Insulin Resistance in Type 2 Diabetes.
Cell Metab
; 32(5): 844-859.e5, 2020 11 03.
Article
in English
| MEDLINE | ID: mdl-32888406
5.
Insulin Resistance in Human iPS Cells Reduces Mitochondrial Size and Function.
Sci Rep
; 6: 22788, 2016 Mar 07.
Article
in English
| MEDLINE | ID: mdl-26948272
6.
Age related skeletal muscle atrophy and upregulation of autophagy in dogs.
Vet J
; 206(1): 54-60, 2015 Oct.
Article
in English
| MEDLINE | ID: mdl-26257260
7.
Genetic insulin resistance is a potent regulator of gene expression and proliferation in human iPS cells.
Diabetes
; 63(12): 4130-42, 2014 Dec.
Article
in English
| MEDLINE | ID: mdl-25059784
8.
Expression of Concern. Prep1 Controls Insulin Glucoregulatory Function in Liver by Transcriptional Targeting of SHP1 Tyrosine Phosphatase. Diabetes 2011;60:138-147. DOI: 10.2337/db10-0860. PMID: 20864515.
Diabetes
; 67(2): 346-347, 2018 02.
Article
in English
| MEDLINE | ID: mdl-29109244
9.
Prep1 controls insulin glucoregulatory function in liver by transcriptional targeting of SHP1 tyrosine phosphatase.
Diabetes
; 60(1): 138-47, 2011 01.
Article
in English
| MEDLINE | ID: mdl-20864515
10.
Prep1 deficiency induces protection from diabetes and increased insulin sensitivity through a p160-mediated mechanism.
Mol Cell Biol
; 28(18): 5634-45, 2008 Sep.
Article
in English
| MEDLINE | ID: mdl-18644868
11.
Phorbol esters induce intracellular accumulation of the anti-apoptotic protein PED/PEA-15 by preventing ubiquitinylation and proteasomal degradation.
J Biol Chem
; 282(12): 8648-57, 2007 Mar 23.
Article
in English
| MEDLINE | ID: mdl-17227770
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