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1.
EBV BILF1 evolved to downregulate cell surface display of a wide range of HLA class I molecules through their cytoplasmic tail.
J Immunol
; 190(4): 1672-84, 2013 Feb 15.
Article
in English
| MEDLINE | ID: mdl-23315076
2.
A CD8+ T cell immune evasion protein specific to Epstein-Barr virus and its close relatives in Old World primates.
J Exp Med
; 204(8): 1863-73, 2007 Aug 06.
Article
in English
| MEDLINE | ID: mdl-17620360
3.
The "Bridge" in the Epstein-Barr virus alkaline exonuclease protein BGLF5 contributes to shutoff activity during productive infection.
J Virol
; 86(17): 9175-87, 2012 Sep.
Article
in English
| MEDLINE | ID: mdl-22696660
4.
EBV lytic-phase protein BGLF5 contributes to TLR9 downregulation during productive infection.
J Immunol
; 186(3): 1694-702, 2011 Feb 01.
Article
in English
| MEDLINE | ID: mdl-21191071
5.
Specific targeting of the EBV lytic phase protein BNLF2a to the transporter associated with antigen processing results in impairment of HLA class I-restricted antigen presentation.
J Immunol
; 182(4): 2313-24, 2009 Feb 15.
Article
in English
| MEDLINE | ID: mdl-19201886
6.
The DNase of gammaherpesviruses impairs recognition by virus-specific CD8+ T cells through an additional host shutoff function.
J Virol
; 82(5): 2385-93, 2008 Mar.
Article
in English
| MEDLINE | ID: mdl-18094150
7.
Rat cytomegalovirus induces a temporal downregulation of major histocompatibility complex class I cell surface expression.
Viral Immunol
; 18(4): 607-15, 2005.
Article
in English
| MEDLINE | ID: mdl-16359227
8.
Host shutoff during productive Epstein-Barr virus infection is mediated by BGLF5 and may contribute to immune evasion.
Proc Natl Acad Sci U S A
; 104(9): 3366-71, 2007 Feb 27.
Article
in English
| MEDLINE | ID: mdl-17360652
9.
Adenovirus-specific CD4+ T cell clones recognizing endogenous antigen inhibit viral replication in vitro through cognate interaction.
J Immunol
; 177(12): 8851-9, 2006 Dec 15.
Article
in English
| MEDLINE | ID: mdl-17142788
10.
Impaired transporter associated with antigen processing-dependent peptide transport during productive EBV infection.
J Immunol
; 174(11): 6829-38, 2005 Jun 01.
Article
in English
| MEDLINE | ID: mdl-15905524
11.
Varicelloviruses avoid T cell recognition by UL49.5-mediated inactivation of the transporter associated with antigen processing.
Proc Natl Acad Sci U S A
; 102(14): 5144-9, 2005 Apr 05.
Article
in English
| MEDLINE | ID: mdl-15793001
12.
Epstein-Barr virus gp42 is posttranslationally modified to produce soluble gp42 that mediates HLA class II immune evasion.
J Virol
; 79(2): 841-52, 2005 Jan.
Article
in English
| MEDLINE | ID: mdl-15613312
13.
Amino acid composition of alpha1/alpha2 domains and cytoplasmic tail of MHC class I molecules determine their susceptibility to human cytomegalovirus US11-mediated down-regulation.
Eur J Immunol
; 33(6): 1707-16, 2003 Jun.
Article
in English
| MEDLINE | ID: mdl-12778489
14.
Human cytomegalovirus-encoded US2 differentially affects surface expression of MHC class I locus products and targets membrane-bound, but not soluble HLA-G1 for degradation.
J Immunol
; 171(12): 6757-65, 2003 Dec 15.
Article
in English
| MEDLINE | ID: mdl-14662880
15.
Signaling mechanisms coupled to tyrosines in the granulocyte colony-stimulating factor receptor orchestrate G-CSF-induced expansion of myeloid progenitor cells.
Blood
; 101(7): 2584-90, 2003 Apr 01.
Article
in English
| MEDLINE | ID: mdl-12468431
16.
Interference with T cell receptor-HLA-DR interactions by Epstein-Barr virus gp42 results in reduced T helper cell recognition.
Proc Natl Acad Sci U S A
; 100(20): 11583-8, 2003 Sep 30.
Article
in English
| MEDLINE | ID: mdl-14504389
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