Protein disulfide isomerase mediates glutathione depletion-induced cytotoxicity.

Okada, Kazushi; Fukui, Masayuki; Zhu, Bao-Ting

Okada, Kazushi; Fukui, Masayuki; Zhu, Bao-Ting.

Afiliación

Okada K; University of Kansas Medical Center, Department of Pharmacology, Toxicology and Therapeutics, Kansas City, 66160, USA; South University of Science and Technology of China, Department of Biology, Shenzhen, 518055, China.
Fukui M; University of Kansas Medical Center, Department of Pharmacology, Toxicology and Therapeutics, Kansas City, 66160, USA.
Zhu BT; University of Kansas Medical Center, Department of Pharmacology, Toxicology and Therapeutics, Kansas City, 66160, USA; South University of Science and Technology of China, Department of Biology, Shenzhen, 518055, China; The Chinese University of Hong Kong (Shenzhen), School of Science and Engineering, Division of Biomedicine and Bioengineering, Shenzhen, 518172, China. Electronic address: BTZhu@CUHK.edu.cn.

Biochem Biophys Res Commun ; 477(3): 495-502, 2016 08 26.

Article en En | MEDLINE | ID: mdl-27317486

Asunto(s)

Glutatión/metabolismo; Proteína Disulfuro Isomerasas/metabolismo; Animales; Especies de Nitrógeno Reactivo/metabolismo; Especies Reactivas de Oxígeno/metabolismo

Palabras clave

Glutamate toxicity; Glutathione; Nitric oxide; Protein disulfide isomerase; Reactive oxygen species; S-nitrosylation

Texto completo

Imprimir

XML

PubMed Links

Buscar en Google

Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Proteína Disulfuro Isomerasas / Glutatión Límite: Animals Idioma: En Revista: Biochem Biophys Res Commun Año: 2016 Tipo del documento: Article País de afiliación: China

Texto completo

Imprimir

XML

PubMed Links

Buscar en Google