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The creatine kinase pathway is a metabolic vulnerability in EVI1-positive acute myeloid leukemia.
Fenouille, Nina; Bassil, Christopher F; Ben-Sahra, Issam; Benajiba, Lina; Alexe, Gabriela; Ramos, Azucena; Pikman, Yana; Conway, Amy S; Burgess, Michael R; Li, Qing; Luciano, Frédéric; Auberger, Patrick; Galinsky, Ilene; DeAngelo, Daniel J; Stone, Richard M; Zhang, Yi; Perkins, Archibald S; Shannon, Kevin; Hemann, Michael T; Puissant, Alexandre; Stegmaier, Kimberly.
Afiliación
  • Fenouille N; Koch Institute for Integrative Cancer Research at Massachusetts Institute of Technology, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
  • Bassil CF; Department of Pediatric Oncology, Dana-Farber Cancer Institute and Boston Children's Hospital, Harvard Medical School, Boston, Massachusetts, USA.
  • Ben-Sahra I; Department of Genetics and Complex Diseases, Harvard School of Public Health, Boston, Massachusetts, USA.
  • Benajiba L; Department of Pediatric Oncology, Dana-Farber Cancer Institute and Boston Children's Hospital, Harvard Medical School, Boston, Massachusetts, USA.
  • Alexe G; Department of Pediatric Oncology, Dana-Farber Cancer Institute and Boston Children's Hospital, Harvard Medical School, Boston, Massachusetts, USA.
  • Ramos A; Broad Institute of Harvard University and Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
  • Pikman Y; Bioinformatics Graduate Program, Boston University, Boston, Massachusetts, USA.
  • Conway AS; Koch Institute for Integrative Cancer Research at Massachusetts Institute of Technology, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
  • Burgess MR; Department of Pediatric Oncology, Dana-Farber Cancer Institute and Boston Children's Hospital, Harvard Medical School, Boston, Massachusetts, USA.
  • Li Q; Department of Pediatric Oncology, Dana-Farber Cancer Institute and Boston Children's Hospital, Harvard Medical School, Boston, Massachusetts, USA.
  • Luciano F; Department of Medicine, University of California San Francisco, San Francisco, California, USA.
  • Auberger P; Internal Medicine Hematology-Oncology, University of Michigan, Ann Arbor, Michigan, USA.
  • Galinsky I; Université Côte d'Azur, UMR INSERM U1065, C3M, Nice, France.
  • DeAngelo DJ; Université Côte d'Azur, UMR INSERM U1065, C3M, Nice, France.
  • Stone RM; Department of Medical Oncology, Dana-Farber Cancer Institute, Harvard Medical School, Boston, Massachusetts, USA.
  • Zhang Y; Department of Medical Oncology, Dana-Farber Cancer Institute, Harvard Medical School, Boston, Massachusetts, USA.
  • Perkins AS; Department of Medical Oncology, Dana-Farber Cancer Institute, Harvard Medical School, Boston, Massachusetts, USA.
  • Shannon K; Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, New York, New York, USA.
  • Hemann MT; Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, New York, New York, USA.
  • Puissant A; Department of Pediatrics and Helen Diller Family Comprehensive Cancer Center, University of California San Francisco, San Francisco, California, USA.
  • Stegmaier K; Koch Institute for Integrative Cancer Research at Massachusetts Institute of Technology, Massachusetts Institute of Technology, Cambridge, Massachusetts, USA.
Nat Med ; 23(3): 301-313, 2017 Mar.
Article en En | MEDLINE | ID: mdl-28191887
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Factores de Transcripción / Proto-Oncogenes / Leucemia Mieloide Aguda / Regulación Neoplásica de la Expresión Génica / Creatina Quinasa / Proteínas de Unión al ADN / Subunidad alfa 2 del Factor de Unión al Sitio Principal Tipo de estudio: Prognostic_studies Límite: Adult / Aged / Aged80 / Female / Humans / Male / Middle aged Idioma: En Revista: Nat Med Asunto de la revista: BIOLOGIA MOLECULAR / MEDICINA Año: 2017 Tipo del documento: Article País de afiliación: Estados Unidos
Texto completo
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Factores de Transcripción / Proto-Oncogenes / Leucemia Mieloide Aguda / Regulación Neoplásica de la Expresión Génica / Creatina Quinasa / Proteínas de Unión al ADN / Subunidad alfa 2 del Factor de Unión al Sitio Principal Tipo de estudio: Prognostic_studies Límite: Adult / Aged / Aged80 / Female / Humans / Male / Middle aged Idioma: En Revista: Nat Med Asunto de la revista: BIOLOGIA MOLECULAR / MEDICINA Año: 2017 Tipo del documento: Article País de afiliación: Estados Unidos