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Non-muscle myosin IIB (Myh10) is required for epicardial function and coronary vessel formation during mammalian development.
Ridge, Liam A; Mitchell, Karen; Al-Anbaki, Ali; Shaikh Qureshi, Wasay Mohiuddin; Stephen, Louise A; Tenin, Gennadiy; Lu, Yinhui; Lupu, Irina-Elena; Clowes, Christopher; Robertson, Abigail; Barnes, Emma; Wright, Jayne A; Keavney, Bernard; Ehler, Elisabeth; Lovell, Simon C; Kadler, Karl E; Hentges, Kathryn E.
Afiliación
  • Ridge LA; Division of Evolution and Genome Sciences, School of Biological Sciences, Faculty of Biology, Medicine, and Health, Manchester Academic Health Science Centre, University of Manchester, Manchester, United Kingdom.
  • Mitchell K; Division of Evolution and Genome Sciences, School of Biological Sciences, Faculty of Biology, Medicine, and Health, Manchester Academic Health Science Centre, University of Manchester, Manchester, United Kingdom.
  • Al-Anbaki A; Division of Evolution and Genome Sciences, School of Biological Sciences, Faculty of Biology, Medicine, and Health, Manchester Academic Health Science Centre, University of Manchester, Manchester, United Kingdom.
  • Shaikh Qureshi WM; Division of Evolution and Genome Sciences, School of Biological Sciences, Faculty of Biology, Medicine, and Health, Manchester Academic Health Science Centre, University of Manchester, Manchester, United Kingdom.
  • Stephen LA; Division of Evolution and Genome Sciences, School of Biological Sciences, Faculty of Biology, Medicine, and Health, Manchester Academic Health Science Centre, University of Manchester, Manchester, United Kingdom.
  • Tenin G; Division of Evolution and Genome Sciences, School of Biological Sciences, Faculty of Biology, Medicine, and Health, Manchester Academic Health Science Centre, University of Manchester, Manchester, United Kingdom.
  • Lu Y; Wellcome Trust Centre for Cell-Matrix Research, Division of Cell-Matrix Biology and Regenerative Medicine, Faculty of Biology, Medicine, and Health, Manchester Academic Health Science Centre, University of Manchester, Manchester, United Kingdom.
  • Lupu IE; Division of Evolution and Genome Sciences, School of Biological Sciences, Faculty of Biology, Medicine, and Health, Manchester Academic Health Science Centre, University of Manchester, Manchester, United Kingdom.
  • Clowes C; Division of Evolution and Genome Sciences, School of Biological Sciences, Faculty of Biology, Medicine, and Health, Manchester Academic Health Science Centre, University of Manchester, Manchester, United Kingdom.
  • Robertson A; Division of Evolution and Genome Sciences, School of Biological Sciences, Faculty of Biology, Medicine, and Health, Manchester Academic Health Science Centre, University of Manchester, Manchester, United Kingdom.
  • Barnes E; Syngenta Ltd, Jealott's Hill International Research Centre, Bracknell, United Kingdom.
  • Wright JA; Syngenta Ltd, Jealott's Hill International Research Centre, Bracknell, United Kingdom.
  • Keavney B; Division of Cardiovascular Sciences, School of Medical Sciences, Faculty of Biology, Medicine, and Health, Manchester Academic Health Science Centre, University of Manchester, Manchester, United Kingdom.
  • Ehler E; Manchester Heart Centre, Central Manchester University Hospitals NHS Foundation Trust, Manchester, United Kingdom.
  • Lovell SC; The Randall Division of Cell and Molecular Biophysics and the Cardiovascular Division, Kings College London, London, United Kingdom.
  • Kadler KE; Division of Evolution and Genome Sciences, School of Biological Sciences, Faculty of Biology, Medicine, and Health, Manchester Academic Health Science Centre, University of Manchester, Manchester, United Kingdom.
  • Hentges KE; Wellcome Trust Centre for Cell-Matrix Research, Division of Cell-Matrix Biology and Regenerative Medicine, Faculty of Biology, Medicine, and Health, Manchester Academic Health Science Centre, University of Manchester, Manchester, United Kingdom.
PLoS Genet ; 13(10): e1007068, 2017 Oct.
Article en En | MEDLINE | ID: mdl-29084269
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Pericardio / Cadenas Pesadas de Miosina / Vasos Coronarios / Miosina Tipo IIB no Muscular / Desarrollo Embrionario Límite: Animals / Humans Idioma: En Revista: PLoS Genet Asunto de la revista: GENETICA Año: 2017 Tipo del documento: Article País de afiliación: Reino Unido
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Pericardio / Cadenas Pesadas de Miosina / Vasos Coronarios / Miosina Tipo IIB no Muscular / Desarrollo Embrionario Límite: Animals / Humans Idioma: En Revista: PLoS Genet Asunto de la revista: GENETICA Año: 2017 Tipo del documento: Article País de afiliación: Reino Unido