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Type I IFN signaling blockade by a PASylated antagonist during chronic SIV infection suppresses specific inflammatory pathways but does not alter T cell activation or virus replication.
Nganou-Makamdop, Krystelle; Billingsley, James M; Yaffe, Zachary; O'Connor, Gregory; Tharp, Gregory K; Ransier, Amy; Laboune, Farida; Matus-Nicodemos, Rodrigo; Lerner, Andrea; Gharu, Lavina; Robertson, Jennifer M; Ford, Mandy L; Schlapschy, Martin; Kuhn, Nadine; Lensch, Alexandra; Lifson, Jeffrey; Nason, Martha; Skerra, Arne; Schreiber, Gideon; Bosinger, Steven E; Douek, Daniel C.
Afiliación
  • Nganou-Makamdop K; Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health, Bethesda, Maryland, United States of America.
  • Billingsley JM; Division of Microbiology and Immunology, Emory Vaccine Center, Yerkes National Primate Research Center, Atlanta, Georgia, United States of America.
  • Yaffe Z; Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health, Bethesda, Maryland, United States of America.
  • O'Connor G; Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health, Bethesda, Maryland, United States of America.
  • Tharp GK; Division of Microbiology and Immunology, Emory Vaccine Center, Yerkes National Primate Research Center, Atlanta, Georgia, United States of America.
  • Ransier A; Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health, Bethesda, Maryland, United States of America.
  • Laboune F; Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health, Bethesda, Maryland, United States of America.
  • Matus-Nicodemos R; Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health, Bethesda, Maryland, United States of America.
  • Lerner A; Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health, Bethesda, Maryland, United States of America.
  • Gharu L; Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health, Bethesda, Maryland, United States of America.
  • Robertson JM; Department of Surgery and Emory Transplant Center, Emory University School of Medicine and Emory Healthcare, Atlanta, GA.
  • Ford ML; Department of Surgery and Emory Transplant Center, Emory University School of Medicine and Emory Healthcare, Atlanta, GA.
  • Schlapschy M; Lehrstuhl für Biologische Chemie, Technische Universität München, Freising (Weihenstephan), Germany.
  • Kuhn N; Lehrstuhl für Biologische Chemie, Technische Universität München, Freising (Weihenstephan), Germany.
  • Lensch A; Lehrstuhl für Biologische Chemie, Technische Universität München, Freising (Weihenstephan), Germany.
  • Lifson J; AIDS and Cancer Virus Program, Frederick National Laboratory for Cancer Research, Frederick, Maryland, United States of America.
  • Nason M; Biostatistics Research Branch, Division of Clinical Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, United States of America.
  • Skerra A; Lehrstuhl für Biologische Chemie, Technische Universität München, Freising (Weihenstephan), Germany.
  • Schreiber G; XL-protein GmbH, Freising, Germany.
  • Bosinger SE; Department of Biomolecular Sciences, Weizmann Institute of Science, Rehovot, Israel.
  • Douek DC; Division of Microbiology and Immunology, Emory Vaccine Center, Yerkes National Primate Research Center, Atlanta, Georgia, United States of America.
PLoS Pathog ; 14(8): e1007246, 2018 08.
Article en En | MEDLINE | ID: mdl-30142226
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Replicación Viral / Linfocitos T / Interferón Tipo I / Síndrome de Inmunodeficiencia Adquirida del Simio / Antirretrovirales / Inflamación Límite: Animals Idioma: En Revista: PLoS Pathog Año: 2018 Tipo del documento: Article País de afiliación: Estados Unidos
Texto completo
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Replicación Viral / Linfocitos T / Interferón Tipo I / Síndrome de Inmunodeficiencia Adquirida del Simio / Antirretrovirales / Inflamación Límite: Animals Idioma: En Revista: PLoS Pathog Año: 2018 Tipo del documento: Article País de afiliación: Estados Unidos