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Tranexamic acid suppresses the release of mitochondrial DNA, protects the endothelial monolayer and enhances oxidative phosphorylation.
Prudovsky, Igor; Carter, Damien; Kacer, Doreen; Palmeri, Monica; Soul, Tee; Kumpel, Chloe; Pyburn, Kathleen; Barrett, Karyn; DeMambro, Victoria; Alexandrov, Ilya; Brandina, Irina; Kramer, Robert; Rappold, Joseph.
Afiliación
  • Prudovsky I; Maine Medical Center Research Institute, Maine Medical Center, Scarborough, Maine.
  • Carter D; Maine Medical Center Research Institute, Maine Medical Center, Scarborough, Maine.
  • Kacer D; Maine Medical Center Department of Trauma, Maine Medical Center, Portland, Maine.
  • Palmeri M; Maine Medical Center Research Institute, Maine Medical Center, Scarborough, Maine.
  • Soul T; Maine Medical Center Research Institute, Maine Medical Center, Scarborough, Maine.
  • Kumpel C; Maine Medical Center Cardiovascular Institute, Maine Medical Center, Portland, Maine.
  • Pyburn K; Maine Medical Center Research Institute, Maine Medical Center, Scarborough, Maine.
  • Barrett K; Maine Medical Center Research Institute, Maine Medical Center, Scarborough, Maine.
  • DeMambro V; Maine Medical Center Research Institute, Maine Medical Center, Scarborough, Maine.
  • Alexandrov I; Maine Medical Center Research Institute, Maine Medical Center, Scarborough, Maine.
  • Brandina I; Maine Medical Center Research Institute, Maine Medical Center, Scarborough, Maine.
  • Kramer R; ActivSignal Company, Natick, Massachusetts.
  • Rappold J; ActivSignal Company, Natick, Massachusetts.
J Cell Physiol ; 234(11): 19121-19129, 2019 11.
Article en En | MEDLINE | ID: mdl-30941770

Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Ácido Tranexámico / Heridas y Lesiones / ADN Mitocondrial / Hemorragia Límite: Animals / Humans Idioma: En Revista: J Cell Physiol Año: 2019 Tipo del documento: Article

Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Ácido Tranexámico / Heridas y Lesiones / ADN Mitocondrial / Hemorragia Límite: Animals / Humans Idioma: En Revista: J Cell Physiol Año: 2019 Tipo del documento: Article