Evolution of MET and NRAS gene amplification as acquired resistance mechanisms in EGFR mutant NSCLC.

Peters, T L; Patil, T; Le, A T; Davies, K D; Brzeskiewicz, P M; Nijmeh, H; Bao, L; Camidge, D R; Aisner, D L; Doebele, R C

Peters, T L; Patil, T; Le, A T; Davies, K D; Brzeskiewicz, P M; Nijmeh, H; Bao, L; Camidge, D R; Aisner, D L; Doebele, R C.

Afiliación

Peters TL; Department of Medicine, Division of Medical Oncology, University of Colorado- Anschutz Medical Campus, Aurora, CO, USA.
Patil T; Department of Medicine, Division of Medical Oncology, University of Colorado- Anschutz Medical Campus, Aurora, CO, USA.
Le AT; Department of Medicine, Division of Medical Oncology, University of Colorado- Anschutz Medical Campus, Aurora, CO, USA.
Davies KD; Department of Pathology, University of Colorado- Anschutz Medical Campus, Aurora, CO, USA.
Brzeskiewicz PM; Department of Pathology, University of Colorado- Anschutz Medical Campus, Aurora, CO, USA.
Nijmeh H; Department of Pathology, University of Colorado- Anschutz Medical Campus, Aurora, CO, USA.
Bao L; Department of Pathology, University of Colorado- Anschutz Medical Campus, Aurora, CO, USA.
Camidge DR; Department of Medicine, Division of Medical Oncology, University of Colorado- Anschutz Medical Campus, Aurora, CO, USA.
Aisner DL; Department of Pathology, University of Colorado- Anschutz Medical Campus, Aurora, CO, USA.
Doebele RC; Department of Medicine, Division of Medical Oncology, University of Colorado- Anschutz Medical Campus, Aurora, CO, USA. robert.doebele@cuanschutz.edu.

NPJ Precis Oncol ; 5(1): 91, 2021 Oct 12.

Article en En | MEDLINE | ID: mdl-34642436

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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Idioma: En Revista: NPJ Precis Oncol Año: 2021 Tipo del documento: Article País de afiliación: Estados Unidos

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