Expression of histone deacetylase (HDAC) family members in bortezomib-refractory multiple myeloma and modulation by panobinostat.

Cheng, Tiewei; Kiser, Kendall; Grasse, Leslie; Iles, Lakesla; Bartholomeusz, Geoffrey; Samaniego, Felipe; Orlowski, Robert Z; Chandra, Joya

Cheng, Tiewei; Kiser, Kendall; Grasse, Leslie; Iles, Lakesla; Bartholomeusz, Geoffrey; Samaniego, Felipe; Orlowski, Robert Z; Chandra, Joya.

Afiliación

Cheng T; Department of Pediatrics Research, University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA.
Kiser K; McGovern Medical School, University of Texas Health Science Center at Houston, Houston, TX 77030, USA.
Grasse L; Department of Pediatrics Research, University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA.
Iles L; Department of Experimental Therapeutics, University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA.
Bartholomeusz G; Department of Experimental Therapeutics, University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA.
Samaniego F; Department of Lymphoma and Myeloma, University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA.
Orlowski RZ; Department of Experimental Therapeutics, University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA.
Chandra J; Department of Lymphoma and Myeloma, University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA.

Cancer Drug Resist ; 4(4): 888-902, 2021.

Article en En | MEDLINE | ID: mdl-34888496

Palabras clave

HDAC6; HDAC7; Histone deacetylase; bortezomib resistance; selective HDAC inhibitors

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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Tipo de estudio: Prognostic_studies Idioma: En Revista: Cancer Drug Resist Año: 2021 Tipo del documento: Article País de afiliación: Estados Unidos

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