Aberrant protein S-nitrosylation contributes to hyperexcitability-induced synaptic damage in Alzheimer's disease: Mechanistic insights and potential therapies.

Ghatak, Swagata; Nakamura, Tomohiro; Lipton, Stuart A

Ghatak, Swagata; Nakamura, Tomohiro; Lipton, Stuart A.

Afiliación

Ghatak S; School of Biological Sciences, National Institute of Science Education and Research, Bhubaneswar, India.
Nakamura T; Neurodegeneration New Medicines Center and Department of Molecular Medicine, The Scripps Research Institute, La Jolla, CA, United States.
Lipton SA; Neurodegeneration New Medicines Center and Department of Molecular Medicine, The Scripps Research Institute, La Jolla, CA, United States.

Front Neural Circuits ; 17: 1099467, 2023.

Article en En | MEDLINE | ID: mdl-36817649

Asunto(s)

Enfermedad de Alzheimer; Humanos; Anciano; Proteína S; Neuronas/fisiología; Especies de Nitrógeno Reactivo

Palabras clave

Alzheimer's disease; NMDA receptors; NitroSynapsin; S-nitrosylation; glutamate excitotoxicity; hyperexcitability

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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Enfermedad de Alzheimer Límite: Aged / Humans Idioma: En Revista: Front Neural Circuits Año: 2023 Tipo del documento: Article País de afiliación: India

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