RESUMO
Introdução: A Síndrome de apnéia e/ou hipopnéia obstrutiva do sono (SAHOS) é caracterizada pela obstrução repetida das vias aéreas ou redução da respiração durante o sono. Esta síndrome é causada por colapso faríngeo contra aumento do esforço espiratório durante o sono. A SAHOS leva à deterioração da qualidade do sono associado à queixa de sonolência diurna excessiva e a efeitos desfavoráveis no sistema cardiovascular. Características orofaciais como retrognatismo mandibular e/ou associado a retrognatismo maxilar, má oclusão de Classe II, com ou sem mordida aberta, e diminuição das vias aéreas orofaríngeas fazem parte do seu quadro. Objetivo: Relato do tratamento ortocirúrgico de um paciente com 31 anos de idade com caraterísticas de SAHOS, má oclusão de Classe II, deficiência mandibular, mordida cruzada posterior e aberta anterior, diminuição da altura facial anteroinferior, aprofundamento do sulco mentolabial, pouca exposição do lábio inferior e nítido desequilíbrio facial. Relato e Resultados: Tratamentos não invasivos para SAHOS passam pelos dispositivos de pressão nasal contínua positiva nas vias aéreas ou aparelhos intra-bucais com o objetivo de manter a mandíbula em posição anteriorizada durante o sono. Nos tratamentos invasivos, a cirurgia de avanço mandibular evita a rotação no sentido anti-horário do complexo maxilomandibular com aumento do espaço orofaríngeo das vias aéreas médias e inferiores. Nas situações com grande impacto esquelético dentofacial associado ao grau de obstrução das vias aéreas será necessário um tratamento descompensatório ortocirúrgico, permitindo assim um restabelecimento respiratório e estético da face. Conclusão: O tratamento descompensatório com avanço cirúrgico bimaxilar foi responsável pela correção da má oclusão e aumento do espaço aéreo, contemplando a expectativa respiratória, funcional e estética.
Introduction: Obstructive sleep apnea and/or hypopnea syndrome (OSAHS) is characterized by repeated obstruction of the airway or reduction of breathing during sleep. This syndrome is caused by pharyngeal collapse against increased respiratory effort during sleep. SAHOS leads to deterioration of sleep quality associated with clinical complaints of excessive daytime sleepiness and unfavorable effects on the cardiovascular system. Orofacial features such as mandibular retrognathism and/or associated maxillary retrognathism, Class II malocclusion, with or without open bite and reduction of the oropharyngeal airway characterize this syndrome. Objective: It is reported an orthosurgical treatment of a 31 years old male patient, with OSAHS characteristics, Class II malocclusion, mandibular deficiency, posterior crossbite and anterior open bite, lower anterior facial height decrease, deepening of the labial sulcus, little exposure of the lower lip and unbalanced face. Report and Results: Non-invasive treatments for OSAHS pass through positive continuous nasal pressure devices in the airways or intra-oral appliances in order to maintain the mandible in an anterior position during sleep. In the invasive treatments protocol, mandibular advancement surgery prevents rotation in the anti-clockwise direction of the maxillomandibular complex with an increase in the oropharyngeal space of the middle and lower airways. In cases with great skeletal dentofacial impact associated with the degree of obstruction of the airway will be necessary an ortho-surgical decompensatory treatment, to allow a respiratory and aesthetic restoration of the face. Conclusion: The decompensatory treatment with bimaxillary surgical advancement corrected the malocclusion, increasing the air space contemplating the respiratory, functional and aesthetic expectation.
Assuntos
Má Oclusão Classe II de Angle , Apneia Obstrutiva do SonoRESUMO
Cleidocranial dysostosis (CCD) is a congenital skeletal disorder with significant manifestations in facial and dental development. Patients are affected with CCD present maxillary deficiency, late dental eruption, and supernumerary teeth. Early and multidisciplinary approach is necessary to treat CCD patients, especially to manage dental eruption and Class III malocclusion with maxillary deficiency. Several orthodontic and surgical interventions are performed to enable traction and extraction of teeth. Yet the maxillary deficiency may be protracted followed by orthodontic dental compensation. On the other hand, it is important to note that CCD patients' treatment is closely related to the severity of transversal and sagittal deformities, as well as the discrepancies in the lower third of the face. In this context, patients with facial impairment highly affected by CCD may need ortho-surgical decompensation to reach more aesthetic outcomes. The present study reports a case of a 14-year-old young patient affected by CCD. Clinically, the patient presented Class III malocclusion, maxillary deficiency, short lower facial third, posterior crossbite, and anterior open bite leading to facial disharmony. The patient underwent treatment in 2 stages: the interceptive approach aimed to transversally expand the maxilla and promote its protraction; and the corrective phase combined with the orthognathic surgery treated the patients' main complains; the anterior open bite, unerupted teeth, and chin prominence. The treatment approach applied in the clinical report allowed the correction of the malocclusion and facial profile satisfying completely the patient's expectations.
