Ankle sensorimotor control and eversion strength after acute ankle inversion injuries.

We performed this study to monitor changes in ankle eversion strength and sensorimotor control functions after acute ankle inversion injury. Forty-four patients with clinical grade II to III first-time ankle inversion sprains were tested for 1) pathologic talar tilt and anterior talar translation at 1 and 12 weeks after injury; 2) isometric eccentric ankle eversion strength and 3) peroneal reaction time to sudden ankle inversion at 3, 6, and 12 weeks after injury; and 4) accuracy of inversion position assessment 1, 3, 6, and 12 weeks after injury. Not all patients could perform all tests at the early follow-up visits. There was mechanical instability in 19 of 40 tested patients at 1 week and in 4 patients at 12 weeks after injury. Eversion strength was 88% of the contralateral side 3 weeks after injury, rising to 96% after 12 weeks. The magnitude of error of inversion position sense was 190% of the contralateral side 1 week after injury; this was still affected after 12 weeks, but fell to 133%. Patients with pathologic talar tilt or anterior talar translation, or both, at 1 week after injury did not show significantly greater error in position assessment or reduction in eversion strength when compared with patients with ankles that remained stable after injury.

[Xanthomonas maltophilia. A cause of epidural abscess in a patient with epidural catheterization]. / Xanthomonas maltophilia. Arsag til epidural absces hos en patient med epiduralkateter.

A case of epidural abscess following continuous spinal epidural catheterization is presented. The clinical signs included spinal ache, root pain, weakness and paralysis but no fever. Xanthomonas maltophilia, an organism closely related to Pseudomonas species, was the causative agent. This has not previously been described as the causative agent of epidural abscesses.