[The enterosorption treatment of patients with acute intestinal infections and chronic colitis with diarrhea]. / Enterosorbtsiine likuvannia khvorykh na hostri kyshkovi infektsiï ta khronichni kolity z diareieiu.

A total of 234 patients with acute intestinal infections (AII) and chronic colitis presenting with diarrhoea were examined. In an acute phase of AII induced by conditionally pathogenic flora, salmonellae and shigellae, as well as in exacerbation of chronic colitis, blood aggregability appears to be on the increase, with the ability of erythrocytes to deformation getting worse. In rectal mucosa hemodynamic disorders are common, manifested by decrease in pulse blood filling, rate of bloodflow predominantly in small and medium-size arteries as well as blood supply as a whole. Applying polysorb to rectal mucosa of such patients with the aid of an atomizer in speedier manner than it is usually done in conventional modes of treatment makes for regression of clinical symptoms of the illness, decreases the level of endotoxicosis, speeds up normalization of blood aggregability, red cell deformability, promoting quick restoration of rectal mucose bloodflow, the anal canal sphincter strength, with its rigidity returning to normal.

[The blood circulation of the rectal mucosa and the functional status of the rectal sphincter in acute infectious enterocolitis]. / Krovobih slyzovoï obolonky priamoï kyshky i funktsional'nyi stan rektal'noho sfinktera pry hostrykh infektsiinykh enterokolitakh.

Significant decrease in the tone of the rectal mucosa venules was to be seen at the climax of acute Proteus and Klebsiella enterocolitis, as evidenced by examinations with the aid of rheorectograph and an analyzer of intracavitary motor activity, general blood supply to the intestinal segment under study being not compromised. The tone of the rectal mucous membrane arterioles is raised at the climax of acute dysentery caused by a Flexner type of organism in erosive and haemorrhagic proctosigmoiditis. With the clinical recovery being set in, the blood supply to this area fails to return to normal. The excitability of the inner anal sphincter was noted to be on the increase at the climax of acute S. flexneri dysentery, this showing up predominantly in erosive and haemorrhagic proctosigmoiditis, ceasing to reveal itself in the period of reconvalescentia.