[Parameters of variation pulsometry, Q-T interval dispersion and frequency of occurrence of late potentials in acute myocardial infarction]. / Parametry variatsionnoi pul'sometrii, dispersiia intervala Q-T i chastota registratsii pozdnikh potentsialov pri ostrom infarkte miokarda.

Registration of standard and high-resolution EGCs, echocardiography in 90 ischemic heart disease (IHD) patients on myocardial infarction week 2 to 6 has demonstrated that deterioration of local myocardial contractility and development of left ventricular dilation increase frequency of late ventricular potentials and worsen parameters of high-resolution ECG. Left ventricular dysfunction is associated with defects in autonomic regulation of cardiac activity. Diminished myocardial contractility had no significant effect on dispersion of repolarisation.

[The use of verapamil for correcting the hemodynamic disorders in the acute period of a myocardial infarct]. / Primenenie verapamila dlia korrektsii narushenii gemodinamiki v ostrom periode infarkta miokarda.

A study of 40 patients with acute myocardial infarction indicates that in those without circulatory insufficiency phynoptin did not reduce cardiac ejection but reduced postloads, energy expenditures and work of the left ventricle myocardium, favoured early stabilization of the cardiovascular system. In cases with cardiac insufficiency phynoptin reduced the contractile capacity of the left ventricle, stasis in the lesser circulation. Associated use of phynoptin and nitrates in acute myocardial infarction with circulatory insufficiency allowed to avoid depressive effect of phynoptin on myocardial contractility and to avoid progression of cardiac insufficiency.

[Drug optimization of the healing of complicated forms of large-focus myocardial infarct]. / Medikamentoznaia optimizatsiia zazhivleniia oslozhnennykh form krupnoochagovogo infarkta miokarda.

The authors give a description of methods of diagnosis and drug optimization of complicated forms of healing of myocardial infarction. It is emphasized that measures of optimization facilitate the clinical course of the disease, reduce the frequency of development of postinfarction aneurysms and further the decrease of lethality.