Assuntos
Febre Hemorrágica com Síndrome Renal/diagnóstico por imagem , Rim/irrigação sanguínea , Síndrome Nefrótica/diagnóstico por imagem , Artéria Renal/diagnóstico por imagem , Veias Renais/diagnóstico por imagem , Adulto , Angiografia , Febre Hemorrágica com Síndrome Renal/patologia , Humanos , Masculino , Síndrome Nefrótica/patologiaAssuntos
Bócio Endêmico/cirurgia , Adolescente , Adulto , Idoso , Criança , Feminino , Bócio Endêmico/patologia , Humanos , Masculino , Pessoa de Meia-Idade , RecidivaAssuntos
Morte , Tuberculose/mortalidade , Adolescente , Adulto , Criança , Pré-Escolar , Humanos , Lactente , Pessoa de Meia-Idade , SibériaRESUMO
Data on morphology of the kidneys in hemorrhagic fever with the renal syndrome (HFRS) are discussed. In the kidneys of 10 decreased patients (with time period from the onset of the disease till death 4--13 days), in whom the disease proceeded with azotemia and oligo-anuria, there were observed oligemia of the glomeruli with secondary changes in the cellular elements, dystrophic changes in the tubules, these changes being particularly pronounced in the proximal segment, and being accompanied by nephrohydrosis; beginning with the 4--6th day signs of regeneration, edema in the walls of vessels and swelling of a hypoxic character were noted. A distinguishing feature of HFRS was a drastic plethora of the jextamedullar zone with hemorrhages due to venous congestion. The changes in the kidneys observed in HFRS were similar to morphological changes in acute renal insufficiency (ARI) of the type of "shock kidney". A characteristic feature of API in HFRS was a high degree of sequestration of the blood flow in the medullar layer.
Assuntos
Febres Hemorrágicas Virais/patologia , Rim/patologia , Adulto , Idoso , Humanos , Glomérulos Renais/patologia , Túbulos Renais/patologia , Pessoa de Meia-IdadeRESUMO
Experiments were conducted on 40 dogs; one lobe of teh thyroid gland or one lobe and 2/3 of the other lobe were removed. There was no gain in weight of the remaining lobe, or of the residue of the gland's lobe. Recovery of the thyroid gland function by hypertrophy of the thyroid epithelium in its remaining portion was achieved by hyperplasia of the intracellular structures.