Effect of orthodontic treatment on alveolar bone thickness in adults: a systematic review.

OBJECTIVES: This review aimed at evaluating changes in alveolar bone thickness after completion of orthodontic treatment. METHODS: Only prospective clinical studies that reported bone thickness in adult patients undergoing non-surgical orthodontic treatment were considered eligible. MEDLINE, EMBASE and LILACS databases were searched for articles published up to July 2018. RESULTS: A total of 12 studies met the selected criteria. Most of the studies showed that orthodontic treatment produces a reduction in bone thickness of incisors, mainly at the palatal side. CONCLUSION: On patients undergoing different orthodontic treatment techniques, there was a significant bone thickness reduction, mainly on the palatal side. CLINICAL RELEVANCE: These findings are relevant and have to be considered in diagnosis and planning of tooth movement, in order to prevent the occurrence of dehiscence and fenestration in alveolar bone.

Rehabilitation treatment associating maxillary atresia and missing superior incisors ­ clinical case study report / Tratamento reabilitador envolvendo atresia maxilar e ausência de incisivos superiores

Multidisciplinary rehabilitating treatment regimens always pose a challenge to orthodontist. The team behind the condition diagnosis chooses a treatment regimen that may achieve predictable and satisfying results both aesthetically and functionally, according to the patient expectation. This article aims to report a regimen that encompasses interaction with dentistry expert areas such as prosthodontics, orthodontics and dental surgery for a patient with maxillary atresia, malocclusion and inferior premolar and two superior incisors missing. The regimen consisted of a rapid surgically-assisted expansion of the maxilla, orthodontic intervention that includes gap closure resulting from two missing incisors through mesialization with prosthesis restoration of the superior teeth.(AU)

Um tratamento reabilitador multidisciplinar é sempre um desafio para o cirurgião-dentista. Uma equipe em conjunto realizando o diagnóstico e plano de tratamento, podem conquistar resultados previsíveis e satisfatórios, tanto estéticos quanto funcionais, respeitando os anseios de cada paciente. O presente trabalho relata um tratamento que engloba interação com as especialidades de prótese dentária, ortodontia e cirurgia, de uma paciente com atresia maxilar, má oclusão e um pré-molar inferior e dois incisivos superiores ausentes. O tratamento realizado consistiu em expansão rápida da maxila assistida cirurgicamente, tratamento ortodôntico incluindo o fechamento dos espaços dos incisivos ausentes através da mesialização dos caninos superiores com restauração e e reabilitação protética dos dentes anterossuperiores. (AU)

Effect of obesity on alveolar bone loss in experimental periodontitis in Wistar rats.

UNLABELLED: Obesity has been linked to higher inflammatory status and periodontal breakdown. OBJECTIVE: The purpose of this study was to investigate the effect of obesity on alveolar bone loss in experimental periodontitis in rats. MATERIAL AND METHODS: Twenty-four female Wistar rats were randomly divided into two groups: obese (n=13), which were fed with "cafeteria diet" (CAF diet--high amounts of sucrose and fat) for 90 days in order to gain weight, and non-obese (n=11) regularly fed rats. Ligature-induced experimental periodontitis was created in all animals. Body weight differed statistically between obese and non-obese groups (277.59 and 223.35 g, respectively) at the moment of the ligature placement. Morphometric registration of alveolar bone loss was carried out after 30 days of ligature placement to determine the effect of obesity on the progression of experimental periodontitis. RESULTS: Intra-group comparisons showed significantly higher alveolar bone loss mean values in maxillary teeth with ligature (P<0.05). Alveolar bone loss [mean (SD), mm] was not statistically different between obese and non-obese groups [0.71 (0.09) and 0.65 (0.07) mm, respectively]. However, when palatal sides are analyzed separately, obese group presented significantly higher alveolar bone loss (P<0.05) as compared to non-obese [0.68 (0.12) and 0.53 (0.13) mm, respectively]. CONCLUSIONS: In spite of the weak differences, it is possible to conclude that the progression of alveolar bone loss in ligature-induced periodontitis can be potentially influenced by body weight in rats.

Effect of obesity on alveolar bone loss in experimental periodontitis in Wistar rats

Obesity has been linked to higher inflammatory status and periodontal breakdown. OBJECTIVE: The purpose of this study was to investigate the effect of obesity on alveolar bone loss in experimental periodontitis in rats. MATERIAL AND METHODS: Twenty-four female Wistar rats were randomly divided into two groups: obese (n=13), which were fed with "cafeteria diet" (CAF diet - high amounts of sucrose and fat) for 90 days in order to gain weight, and non-obese (n=11) regularly fed rats. Ligature-induced experimental periodontitis was created in all animals. Body weight differed statistically between obese and non-obese groups (277.59 and 223.35 g, respectively) at the moment of the ligature placement. Morphometric registration of alveolar bone loss was carried out after 30 days of ligature placement to determine the effect of obesity on the progression of experimental periodontitis. RESULTS: Intra-group comparisons showed significantly higher alveolar bone loss mean values in maxillary teeth with ligature (P<0.05). Alveolar bone loss [mean (SD), mm] was not statistically different between obese and non-obese groups [0.71 (0.09) and 0.65 (0.07) mm, respectively]. However, when palatal sides are analyzed separately, obese group presented significantly higher alveolar bone loss (P<0.05) as compared to non-obese [0.68 (0.12) and 0.53 (0.13) mm, respectively]. CONCLUSIONS: In spite of the weak differences, it is possible to conclude that the progression of alveolar bone loss in ligature-induced periodontitis can be potentially influenced by body weight in rats.