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Whereas point-gap topological phases are responsible for exceptional phenomena intrinsic to non-Hermitian systems, their realization in quantum materials is still elusive. Here, we propose a simple and universal platform of point-gap topological phases constructed from Hermitian topological insulators and superconductors. We show that (d-1)-dimensional point-gap topological phases are realized by making a boundary in d-dimensional topological insulators and superconductors dissipative. A crucial observation of the proposal is that adding a decay constant to boundary modes in d-dimensional topological insulators and superconductors is topologically equivalent to attaching a (d-1)-dimensional point-gap topological phase to the boundary. We furthermore establish the proposal from the extended version of the Nielsen-Ninomiya theorem, relating dissipative gapless modes to point-gap topological numbers. From the bulk-boundary correspondence of the point-gap topological phases, the resultant point-gap topological phases exhibit exceptional boundary states or in-gap higher-order non-Hermitian skin effects.
RESUMO
There is a common belief in the condensed matter community that bulk quantities become insensitive to the boundary condition in the infinite-volume limit. Here we reconsider this statement in terms of recent arguments of non-Hermitian skin effects-strong dependence of spectra on boundary conditions for the non-Hermitian Hamiltonians-in the traditional Green's function formalism. We find the criterion for quantities to be sensitive or insensitive against the boundary condition in Hermitian correlated or disordered systems, which is characterized by the residue theorem. We also discuss the uncertainty of the quasiparticle energy under the skin effects in terms of non-normal pseudospectra, which can be tested via the sharp optical absorption from the bulk-surface coupling. Our result indicates that pseudo quantum number emerges as a consequence of large nonnormality.
RESUMO
A unique feature of non-Hermitian systems is the skin effect, which is the extreme sensitivity to the boundary conditions. Here, we reveal that the skin effect originates from intrinsic non-Hermitian topology. Such a topological origin not merely explains the universal feature of the known skin effect, but also leads to new types of the skin effects-symmetry-protected skin effects. In particular, we discover the Z_{2} skin effect protected by time-reversal symmetry. On the basis of topological classification, we also discuss possible other skin effects in arbitrary dimensions. Our work provides a unified understanding about the bulk-boundary correspondence and the skin effects in non-Hermitian systems.
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Quantum phase transitions are intriguing and fundamental cooperative phenomena in physics. Analyzing a superconducting nanowire with spin-dependent non-Hermitian hopping, we discover a topological quantum phase transition driven by infinitesimal cascade instability. The anomalous phase transition is complementary to the universal non-Bloch wave behavior of non-Hermitian systems. We show that an infinite small magnetic field drastically suppresses the non-Hermitian skin effect, inducing a topological phase with Majorana boundary states. Furthermore, by identifying the bulk topological invariant, we establish the non-Hermitian bulk-boundary correspondence that does not have a Hermitian counterpart. We also discuss an experimental realization of the system by using the spin-current injection to a quantum wire.
RESUMO
We generalize the concept of the spin-momentum locking to magnonic systems and derive the formula to calculate the spin expectation value for one-magnon states of general two-body spin Hamiltonians. We give no-go conditions for magnon spin to be independent of momentum. As examples of the magnon spin-momentum locking, we analyze a one-dimensional antiferromagnet with the Néel order and two-dimensional kagome lattice antiferromagnets with the 120° structure. We find that the magnon spin depends on its momentum even when the Hamiltonian has the z-axis spin rotational symmetry, which can be explained in the context of a singular band point or a U(1) symmetry breaking. A spin vortex in momentum space generated in a kagome lattice antiferromagnet has the winding number Q=-2, while the typical one observed in topological insulator surface states is characterized by Q=+1. A magnonic analogue of the surface states, the Dirac magnon with Q=+1, is found in another kagome lattice antiferromagnet. We also derive the sum rule for Q by using the Poincaré-Hopf index theorem.
RESUMO
BACKGROUND: This study characterized the changes in quality and quantity of saliva, and changes in the salivary metabolomic profile, to understand the effects of masticatory stimulation. METHODS: Stimulated and unstimulated saliva samples were collected from 55 subjects and salivary hydrophilic metabolites were comprehensively quantified using capillary electrophoresis-time-of-flight mass spectrometry. RESULTS: In total, 137 metabolites were identified and quantified. The concentrations of 44 metabolites in stimulated saliva were significantly higher than those in unstimulated saliva. Pathway analysis identified the upregulation of the urea cycle and synthesis and degradation pathways of glycine, serine, cysteine and threonine in stimulated saliva. A principal component analysis revealed that the effect of masticatory stimulation on salivary metabolomic profiles was less dependent on sample population sex, age, and smoking. The concentrations of only 1 metabolite in unstimulated saliva, and of 3 metabolites stimulated saliva, showed significant correlation with salivary secretion volume, indicating that the salivary metabolomic profile and salivary secretion volume were independent factors. CONCLUSIONS: Masticatory stimulation affected not only salivary secretion volume, but also metabolite concentration patterns. A low correlation between the secretion volume and these patterns supports the conclusion that the salivary metabolomic profile may be a new indicator to characterize masticatory stimulation.