[The influence of various levels of carotene and protein in the diet on the index of invasion and digestive enzyme activity in chickens infected with Ascaridia galli]. / Wplyw zróznicowanego poziomu karotenu i bialka w diecie na wskazniki inwazji oraz aktywnosc enzymów trawiennych u kurczat zarazonych Ascaridia galli.

Chickens 10 days old were infected with 500 eggs of Ascaridia galli. They were fed with diet containing 11% or 19% protein. Some of them were given beta-carotene in a dose equivalent to daily requirement of vitamin A, others were given double dose of beta-carotene. The intensity and extensity of invasion, the body weight, and activities of alpha-amylase, lipase and trypsin in pancreas and duodenal content were measured. Application of beta-carotene (especially in a double dose) caused the decrease in the invasion index and moderated the changes in the activity of digestive enzymes, occurring in the infected chickens.

[Effect of vitamin A and protein levels in the diet of chickens infected with Ascaridia galli on activity of digestive enzymes in pancreas and small intestine]. / Wplyw poziomu witaminy a i bialka w diecie kurczat zarazonych nicieniem ascaridia galli na aktywnosc enzymów trawiennych w trzustce i jelicie cienkim.

Hybrid chickens Astra S fed vitamin A deficiency diet containing 11 or 19% protein were infected with 500 invasive eggs of Ascaridia galli. One part of them received vitamin A in a dose corresponding to daily requirement, other part in double dose. After 7 weeks the extensity and intensity of ascaridiosis were determined. In pancreas and intestinal contents the activities of alpha-amylase, lipase and trypsin were measured. The invasion index was the highest in the group of chickens fed the protein and vitamin A deficiency diet. Vitamin A in a requirement dose lowered the parasite index. The results with double dose of vitamin A were worse in comparison with single dose. The activities of digestive enzymes were lower in infected animals than in the control groups. Vitamin A acted as moderator of the enzymatic changes during ascaridiosis.