[Post-respiratory infection reactive arthritis--etiology, clinical and laboratory characteristics and early prognosis]. / Postrespiratornyi reaktivnyi artrit--étiologiia, kliniko-laboratornaia khrakteristika i rannii prognoz.

AIM: To study etiology, clinico-laboratory characteristics and early prognosis of reactive arthritis developing after respiratory infections (priReA). MATERIAL AND METHODS: Blood samples from 69 priReA patients aged 17-45 years were examined for specific antibodies to 28 bacterial and viral infectious agents, and antigens of group HLA-B7-CREG (B7, B13, B22, B27, B40, B73, Cw2). The test were made in hospital and 3 months after the patients' discharge. RESULTS: The priReA respiratory etiology was discovered in 31 patients, urogenital--in 14 cases and enterocolitic one--in 7 patients. Streptococcus and pneumoneal chlamydia cause priReA most frequently. HLA-B40 carriers are more predisposed to priReA. priReA runs with less frequent Reiter's syndrome and lesions of axial and extraarticular structures, better early prognosis. CONCLUSION: In young patients with early ReA respiratory etiology is observed more frequently than urogenital and enterocolitic ones. Compared to other kinds of ReA, priReA demonstrates some genetic, clinical and prognostic peculiarities.

[Cytomorphometric studies on normal stomach mucosa, precancerous diseases and stomach cancer]. / Zytomorphometrische Untersuchungen bei normaler Magenschleimhaut, präkanzerösen Krankheiten und Magenkrebs.

By means of a graduated microscope eyepiece-micrometer cytomorphometric investigations have been carried out on paraffin sections from 120 patients with normal gastric mucosa, gastritis, gastric ulcer, polyps and carcinoma of the stomach. In normal mucosa and gastritis, the number of the cell groups is low, the curves are predominantly single peaked, shifted to the left and the sizes of the nuclei are small. In gastric ulcer, polyps and carcinoma the number of the cell groups is high, the curves are multi-peaked, without any well expressed peaks, shifted to the right and the sizes of the nuclei are large.

[Carcinogenic action of diarsenic trioxide]. / Vurkhu kantserogennoto deistvie na dvuarsenoviia triokis.

Biarsenic trioxide under the experimental conditions outlined in the paper causes mainly adenopapillomatous proliferations in the intestine and partly in the stomach, as well as adenomatous proliferations in the lungs, all of which may be considered as precancerous conditions. Having in mind that no cancerigenic actions of arsenic has been proved hitherto, the experimental data submitted assume a particular theoretical and practical interest.