RESUMO
The behaviour of Salmonella enteritidis during the Feta cheese making process was investigated. Two lots of pasteurized whole ewe's milk were inoculated to contain 10(6) cfu/ml of S. enteritidis (strain AS1 and AS2) and were processed into Feta cheese following standard procedures. All samples were examined for S. enteritidis both quantitatively and qualitatively, while moisture, fat, water activity and pH were also measured. S. enteritidis was enumerated in duplicate samples by surface plating on SS agar. Selected salmonella-like colonies were identified biochemically and serologically. The enumerations have shown that S. enteritidis was initially entrapped in the curd. Then the growth of S. enteritidis gradually decreased and no Salmonellae were quantitatively enumerated after the 23rd day. However S. enteritidis was detected qualitatively in samples taken until the 38th day.
Assuntos
Queijo/microbiologia , Manipulação de Alimentos , Microbiologia de Alimentos , Salmonella enteritidis/crescimento & desenvolvimento , Animais , Contagem de Colônia Microbiana , Concentração de Íons de Hidrogênio , Leite/microbiologia , OvinosRESUMO
The ability of Salmonella enteritidis to grow during the Feta cheese-making process and to survive during ripening and storage of the cheese was investigated. Unpasteurized whole ewe's milk was inoculated to contain 106-107 CFU/ml of S. enteritidis and was processed into Feta cheese following standard procedures. All samples were examined for S. enteritidis both quantitatively and qualitatively, while moisture, fat, water activity, and pH values were also measured. S. enteritidis was enumerated in duplicate samples by surface plating on SS agar. Selected Salmonella colonies were identified biochemically and serologically. The enumerations have shown that S. enteritidis was entrapped in curd with the population increasing to a maximum during the First 48 h. Thereafter, the growth of S. enteritidis was inhibited and surviving cells persisted in the curd for up to 20 d.
RESUMO
A case of parenchymal and intraventricular hemorrhage in a full-term neonate is reported. The underlying cause in our patient is thought to be related to cerebral sinovenous occlusive disease secondary to Protein C deficiency, a rare coagulopathy.