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Genet Mol Res ; 12(4): 5267-77, 2013 Nov 07.
Artigo em Inglês | MEDLINE | ID: mdl-24301787

RESUMO

We examined the effect of microRNAs on 3T3-L1 adipocyte differentiation and expression of adipocyte-specific gene fatty acid-binding protein 4 (FABP4). We screened and identified adipo-related microRNAs during 3T3-L1 adipocyte differentiation with a microRNA microarray. High expression plasmids of miR-24 and miR-21 were constructed and transfected into 3T3-L1 preadipocytes by lipofectamine. The effects of miR-24 and miR-21 on 3T3-L1 adipocyte differentiation were observed, and the protein and mRNA expression levels of FABP4 and AP-1 were determined. The expression profiles of microRNAs significantly changed during 3T3-L1 adipocyte differentiation. The expression of 33 microRNAs was downregulated, among which downregulation of miR-24 was the most extensive. There were 17 microRNAs with upregulated expression; the highest levels were found for miR-21. miR-24 significantly inhibited 3T3-L1 adipocyte differentiation and maturity, while miR-21 had no significant effect. In addition, miR-24 significantly inhibited the expression of FABP4, while it upregulated AP-1 expression, but had no effect on the level of FABP4 mRNA. miR-21 had no effect on FABP4 protein and mRNA expression. AP-1 silencing could, at least partially, reverse the inhibitory effect of miR-24 on FABP4 expression. We conclude that microRNA expression profiles change significantly during 3T3- L1 adipocyte differentiation and that miR-24 plays an important role in regulating adipocyte differentiation and FABP4 expression. The mechanism involved may be the upregulation of AP-1.


Assuntos
Adipócitos/citologia , Adipócitos/metabolismo , Diferenciação Celular/genética , Proteínas de Ligação a Ácido Graxo/genética , Regulação da Expressão Gênica , MicroRNAs/genética , Células 3T3-L1 , Animais , Proteínas de Ligação a Ácido Graxo/metabolismo , Perfilação da Expressão Gênica , Camundongos , Interferência de RNA , RNA Mensageiro/genética , RNA Mensageiro/metabolismo , Fator de Transcrição AP-1/genética , Fator de Transcrição AP-1/metabolismo
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