RESUMO
The basic clinical signs of calcifications of the basal ganglia and cerebellar dentate nuclei are discussed with separation of various forms of that disease. Particular attention was given to the factors which might predispose to the development of the so called idiopathic calcifications (Fahr syndrome), and secondary calcifications in various forms of parathyroid hypofunction.
Assuntos
Gânglios da Base/patologia , Encefalopatias/patologia , Calcinose/patologia , Núcleos Cerebelares/patologia , Síndrome , Adolescente , Adulto , Encefalopatias/diagnóstico , Encefalopatias/etiologia , Calcinose/etiologia , Criança , Pré-Escolar , Aberrações Cromossômicas , Transtornos Cromossômicos , Feminino , Humanos , Lactente , Masculino , Pessoa de Meia-Idade , Doenças das Paratireoides/complicações , Doenças das Paratireoides/fisiopatologia , Glândulas Paratireoides/fisiopatologiaAssuntos
Veias Hepáticas/anatomia & histologia , Adulto , Idoso , Feminino , Humanos , Masculino , Pessoa de Meia-IdadeAssuntos
Veia Cava Inferior/anatomia & histologia , Adulto , Fatores Etários , Idoso , Estatura , Feminino , Humanos , Masculino , Pessoa de Meia-Idade , Fatores SexuaisRESUMO
The purpose of this study was to analyze the possibilities of collateral venous circulation in the liver of man. 100 specimens taken from autopsy, were injected with vinyl-polychloride and corroded. In spite of opinion of some investigators, it has been found that the hepatic veins often (possibly always) anastomose between each other. The largest are the anastomoses between the right and middle hepatic veins. It is evident that there exist good conditions for the venous collateral circulation between all the hepatic veins in the human liver.
