Denegación de incapacidad versus ineptitud laboral / Denial of disability versus inability to meet essential job functions

El retorno al trabajo tras una baja médica prolongada comporta una valoración tanto de la capacidad laboral como de la aptitud para el trabajo. Nos encontramos con una trabajadora auxiliar administrativa con lesión congénita en cuerdas vocales, a quien se le deniega la incapacidad permanente total. Es valorada en el reconocimiento médico tras ausencia prolongada resultando apta con limitaciones. Tras la revisión de la evaluación de riesgos de su puesto y de todos los compatibles con su categoría profesional se procede a despido objetivo por ineptitud sobrevenida. La valoración de la capacidad/incapacidad laboral debe ser una valoración multidisciplinar, contemplando de forma integrada el diagnóstico, el tratamiento, la evolución, la perspectiva preventiva y la laboral

The return to work process following a prolonged medical leave includes an assessment of both work capacity and ability to meet essential job functions. We present the case of an administrative assistant with a congenital vocal cord injury who was denied total permanent disability. The initial fitness for duty examination after her prolonged absence resulted in a determination of fitness, but with limitations. After performing a risk assessment and review of the essential job functions of her occupation, she was dismissed based on inability to meet the essential demands of the job. Assessments of work capacity/disability must be multidisciplinary, integrating the diagnosis, treatment, course of the illness, and conducted from both a preventive and work perspective

Histopathologic investigations of the unphonated human vocal fold mucosa.

CONCLUSION: Vocal fold vibration (phonation) after birth is one of the important factors in the growth and development of the human vocal fold mucosa. OBJECTIVES: Stellate cells in the maculae flavae located at both ends of the vocal fold mucosa are inferred to be involved in the metabolism of extracellular matrices. Maculae flavae are also considered to be an important structure in the growth and development of the human vocal fold mucosa. Tension caused by phonation (vocal fold vibration) is hypothesized to stimulate stellate cells to accelerate production of extracellular matrices. Vocal fold mucosae unphonated since birth were investigated histologically. SUBJECTS AND METHODS: Vocal fold mucosae, which were unphonated since birth, of three younger adults (17, 24, 28 years old) were investigated by light and electron microscopy. RESULTS: Vocal fold mucosae were hypoplastic and rudimentary and did not have a vocal ligament, Reinke's space or a layered structure. The lamina propria appeared as a uniform structure. Some stellate cells in the maculae flavae showed degeneration. Not many vesicles were present at the periphery of the cytoplasm. The stellate cells synthesized fewer extracellular matrices, such as fibrous protein and glycosaminoglycan. Cytoplasmic processes of the stellate cells were short and shrinking. The stellate cells appeared to have decreased activity.

[Congenital laryngotracheo-esophageal cleft: diagnosis and surgical treatment by anterior, translaryngeal approach]. / Die kongenitale, laryngotracheo-ösophageale Spaltbildung: Diagnostik und operative Behandlung über einen anterioren, translaryngealen Zugang.

BACKGROUND: Laryngotracheoesophageal cleft is a rare but potentially life-threatening anomaly. Less than 200 cases have been published to date. Both the diagnostic and therapeutic recommendations are discussed controversially in the international literature. PATIENT: We report on the diagnostic and surgical management of a type III cleft larynx in a one month old male presenting with aspiration, pneumonia, and aphonia. Hypoplasia of the cricoarytenoid muscles was associated to the cleft. Rigid endoscopy was found to be the best tool for the diagnostic exploration of the cleft, whereas flexible endoscopy failed to detect the defect. The cleft was broadly exposed using a modified anterior translaryngeal approach that included a tracheostomy. After debriding the mucosal margins, the defect was closed in two layers, and a t-shaped Montgomery tube was implanted. Two further revisions using the mentioned translaryngeal approach and one endoscopic procedure were necessary to achieve complete and permanent closure of the cleft. RESULTS: Twenty months after birth the boy is able to swallow thick and liquid food without any problems. Stable scar tissue has grown within the former cleft region. The vocal cords are somewhat thickened but mobile in a reduced range. Both the trachea and the esophagus show quite normal diameters. CONCLUSION: Considering the fact that the arytenoid cartilages touch or overlap each other a congenital defect within the posterior midline of the larynx can only be diagnosed by rigid endoscopy that spreads the cleft apart. In addition to our positive experiences with traumatic fistulas and stenosis of the juvenile trachea we recommend now the anterior vertical laryngeal incision for the operative management of the congenital type III cleft larynx. This direct open approach provides excellent exposure of all components of the defect without the risk of recurrent laryngeal nerve injury. Subglottic stenosis or impaired stability of the larynx described by other authors did not occur in this case. However, the postoperative period is relatively short and careful follow-up for a period of several years is therefore required.

Medullary nuclear dysplasia (Möbius syndrome) in an infant with recurrent apnea and isoelectric EEG.

From birth, an infant girl had repeated episodes of loss of consciousness, apnea and bradycardia, aphonia, and severe pharyngeal-esophageal discoordination. These functional deficits correlated with a gross and microscopic malformation of the medulla oblongata, which is characterized by profound deficits of motor and sensory nuclei and of the reticular formation. We have classified this as an example of Möbius syndrome. No etiology was suggested from the history. Histopathologic findings are compatible with an early massive insult to developing structures in the tegmentum of the medulla oblongata.