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The triggering receptor expressed on myeloid cells 2 inhibits complement component 1q effector mechanisms and exerts detrimental effects during pneumococcal pneumonia.
Sharif, Omar; Gawish, Riem; Warszawska, Joanna M; Martins, Rui; Lakovits, Karin; Hladik, Anastasiya; Doninger, Bianca; Brunner, Julia; Korosec, Ana; Schwarzenbacher, Roland E; Berg, Tiina; Kralovics, Robert; Colinge, Jacques; Mesteri, Ildiko; Gilfillan, Susan; Salmaggi, Andrea; Verschoor, Admar; Colonna, Marco; Knapp, Sylvia.
Afiliação
  • Sharif O; CeMM - Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria; Department of Medicine I, Laboratory of Infection Biology, Medical University of Vienna, Vienna, Austria.
  • Gawish R; CeMM - Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria; Department of Medicine I, Laboratory of Infection Biology, Medical University of Vienna, Vienna, Austria.
  • Warszawska JM; CeMM - Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria; Department of Medicine I, Laboratory of Infection Biology, Medical University of Vienna, Vienna, Austria.
  • Martins R; CeMM - Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria; Department of Medicine I, Laboratory of Infection Biology, Medical University of Vienna, Vienna, Austria.
  • Lakovits K; Department of Medicine I, Laboratory of Infection Biology, Medical University of Vienna, Vienna, Austria.
  • Hladik A; CeMM - Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria; Department of Medicine I, Laboratory of Infection Biology, Medical University of Vienna, Vienna, Austria.
  • Doninger B; CeMM - Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria; Department of Medicine I, Laboratory of Infection Biology, Medical University of Vienna, Vienna, Austria.
  • Brunner J; CeMM - Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria; Department of Medicine I, Laboratory of Infection Biology, Medical University of Vienna, Vienna, Austria.
  • Korosec A; CeMM - Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria; Department of Medicine I, Laboratory of Infection Biology, Medical University of Vienna, Vienna, Austria.
  • Schwarzenbacher RE; CeMM - Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria; Department of Medicine I, Laboratory of Infection Biology, Medical University of Vienna, Vienna, Austria.
  • Berg T; CeMM - Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria.
  • Kralovics R; CeMM - Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria.
  • Colinge J; CeMM - Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria.
  • Mesteri I; Department of Pathology, Medical University of Vienna, Vienna, Austria.
  • Gilfillan S; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, Missouri, United States of America.
  • Salmaggi A; Department of Clinical Neurosciences, Istituto Nazionale Neurologico Carlo Besta, Milano, Italy.
  • Verschoor A; Institute for Medical Microbiology, Immunology and Hygiene, Technical University Munich, Munich, Germany.
  • Colonna M; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, Missouri, United States of America.
  • Knapp S; CeMM - Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria; Department of Medicine I, Laboratory of Infection Biology, Medical University of Vienna, Vienna, Austria.
PLoS Pathog ; 10(6): e1004167, 2014 Jun.
Article em En | MEDLINE | ID: mdl-24945405
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Pneumonia Pneumocócica / Glicoproteínas de Membrana / Receptores Imunológicos / Complemento C1q / Macrófagos Alveolares / Mucosa Respiratória / Modelos Animais de Doenças / Pulmão Limite: Animals Idioma: En Revista: PLoS Pathog Ano de publicação: 2014 Tipo de documento: Article País de afiliação: Áustria País de publicação: Estados Unidos
Texto completo
Adicionar na Minha BVS
Imprimir
XML
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Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Pneumonia Pneumocócica / Glicoproteínas de Membrana / Receptores Imunológicos / Complemento C1q / Macrófagos Alveolares / Mucosa Respiratória / Modelos Animais de Doenças / Pulmão Limite: Animals Idioma: En Revista: PLoS Pathog Ano de publicação: 2014 Tipo de documento: Article País de afiliação: Áustria País de publicação: Estados Unidos