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HIF-1-mediated suppression of acyl-CoA dehydrogenases and fatty acid oxidation is critical for cancer progression.
Huang, De; Li, Tingting; Li, Xinghua; Zhang, Long; Sun, Linchong; He, Xiaoping; Zhong, Xiuying; Jia, Dongya; Song, Libing; Semenza, Gregg L; Gao, Ping; Zhang, Huafeng.
Afiliação
  • Huang; Institute for Cancer Research and CAS Key Laboratory of Innate Immunity and Chronic Disease, Innovation Center for Cell Biology, School of Life Science, University of Science and Technology of China, Hefei 230027, China.
  • Li T; Institute for Cancer Research and CAS Key Laboratory of Innate Immunity and Chronic Disease, Innovation Center for Cell Biology, School of Life Science, University of Science and Technology of China, Hefei 230027, China.
  • Li X; State Key Laboratory of Oncology in Southern China and Departments of Experimental Research, Sun Yat-sen University Cancer Center, Guangzhou 510060, China.
  • Zhang L; Institute for Cancer Research and CAS Key Laboratory of Innate Immunity and Chronic Disease, Innovation Center for Cell Biology, School of Life Science, University of Science and Technology of China, Hefei 230027, China.
  • Sun L; Institute for Cancer Research and CAS Key Laboratory of Innate Immunity and Chronic Disease, Innovation Center for Cell Biology, School of Life Science, University of Science and Technology of China, Hefei 230027, China.
  • He X; Institute for Cancer Research and CAS Key Laboratory of Innate Immunity and Chronic Disease, Innovation Center for Cell Biology, School of Life Science, University of Science and Technology of China, Hefei 230027, China.
  • Zhong X; Institute for Cancer Research and CAS Key Laboratory of Innate Immunity and Chronic Disease, Innovation Center for Cell Biology, School of Life Science, University of Science and Technology of China, Hefei 230027, China.
  • Jia D; Institute for Cancer Research and CAS Key Laboratory of Innate Immunity and Chronic Disease, Innovation Center for Cell Biology, School of Life Science, University of Science and Technology of China, Hefei 230027, China.
  • Song L; State Key Laboratory of Oncology in Southern China and Departments of Experimental Research, Sun Yat-sen University Cancer Center, Guangzhou 510060, China.
  • Semenza GL; Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
  • Gao P; Institute for Cancer Research and CAS Key Laboratory of Innate Immunity and Chronic Disease, Innovation Center for Cell Biology, School of Life Science, University of Science and Technology of China, Hefei 230027, China.
  • Zhang H; Institute for Cancer Research and CAS Key Laboratory of Innate Immunity and Chronic Disease, Innovation Center for Cell Biology, School of Life Science, University of Science and Technology of China, Hefei 230027, China. Electronic address: hzhang22@ustc.edu.cn.
Cell Rep ; 8(6): 1930-1942, 2014 Sep 25.
Article em En | MEDLINE | ID: mdl-25242319
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Acil-CoA Desidrogenase / Acil-CoA Desidrogenase de Cadeia Longa / Subunidade alfa do Fator 1 Induzível por Hipóxia / Ácidos Graxos / Neoplasias Hepáticas Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Revista: Cell Rep Ano de publicação: 2014 Tipo de documento: Article País de afiliação: China País de publicação: Estados Unidos
Texto completo
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Acil-CoA Desidrogenase / Acil-CoA Desidrogenase de Cadeia Longa / Subunidade alfa do Fator 1 Induzível por Hipóxia / Ácidos Graxos / Neoplasias Hepáticas Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Revista: Cell Rep Ano de publicação: 2014 Tipo de documento: Article País de afiliação: China País de publicação: Estados Unidos