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Disturbed flow enhances inflammatory signaling and atherogenesis by increasing thioredoxin-1 level in endothelial cell nuclei.
Go, Young-Mi; Son, Dong Ju; Park, Heonyong; Orr, Michael; Hao, Li; Takabe, Wakako; Kumar, Sandeep; Kang, Dong Won; Kim, Chan Woo; Jo, Hanjoong; Jones, Dean P.
Afiliação
  • Go YM; Division of Pulmonary, Allergy and Critical Care Medicine, Department of Medicine, Emory University, Atlanta, Georgia, United States of America.
  • Son DJ; Division of Cardiology, Department of Medicine, Emory University, Atlanta, Georgia, United States of America.
  • Park H; Department of Molecular Biology, Dankook University, Yongin-si, South Korea.
  • Orr M; Division of Pulmonary, Allergy and Critical Care Medicine, Department of Medicine, Emory University, Atlanta, Georgia, United States of America.
  • Hao L; Division of Pulmonary, Allergy and Critical Care Medicine, Department of Medicine, Emory University, Atlanta, Georgia, United States of America.
  • Takabe W; Division of Cardiology, Department of Medicine, Emory University, Atlanta, Georgia, United States of America.
  • Kumar S; Division of Cardiology, Department of Medicine, Emory University, Atlanta, Georgia, United States of America.
  • Kang DW; Division of Cardiology, Department of Medicine, Emory University, Atlanta, Georgia, United States of America.
  • Kim CW; Division of Cardiology, Department of Medicine, Emory University, Atlanta, Georgia, United States of America.
  • Jo H; Division of Cardiology, Department of Medicine, Emory University, Atlanta, Georgia, United States of America; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University, Atlanta, Georgia, United States of America.
  • Jones DP; Division of Pulmonary, Allergy and Critical Care Medicine, Department of Medicine, Emory University, Atlanta, Georgia, United States of America.
PLoS One ; 9(9): e108346, 2014.
Article em En | MEDLINE | ID: mdl-25265386
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Tiorredoxinas / Artérias Carótidas / Núcleo Celular / Aterosclerose / Células Endoteliais da Veia Umbilical Humana Tipo de estudo: Prognostic_studies Limite: Animals / Humans / Male Idioma: En Revista: PLoS One Assunto da revista: CIENCIA / MEDICINA Ano de publicação: 2014 Tipo de documento: Article País de afiliação: Estados Unidos País de publicação: Estados Unidos
Texto completo
Adicionar na Minha BVS
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XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Tiorredoxinas / Artérias Carótidas / Núcleo Celular / Aterosclerose / Células Endoteliais da Veia Umbilical Humana Tipo de estudo: Prognostic_studies Limite: Animals / Humans / Male Idioma: En Revista: PLoS One Assunto da revista: CIENCIA / MEDICINA Ano de publicação: 2014 Tipo de documento: Article País de afiliação: Estados Unidos País de publicação: Estados Unidos