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IRS-2 Partially Compensates for the Insulin Signal Defects in IRS-1-/- Mice Mediated by miR-33.
Tang, Chen-Yi; Man, Xiao-Fei; Guo, Yue; Tang, Hao-Neng; Tang, Jun; Zhou, Ci-La; Tan, Shu-Wen; Wang, Min; Zhou, Hou-De.
Afiliação
  • Tang CY; Department of Endocrinology and Metabolism, National Clinical Research Center for Metabolic Diseases, The Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China.
  • Man XF; Department of Endocrinology and Metabolism, National Clinical Research Center for Metabolic Diseases, The Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China.
  • Guo Y; Department of Endocrinology and Metabolism, National Clinical Research Center for Metabolic Diseases, The Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China.
  • Tang HN; Department of Endocrinology and Metabolism, National Clinical Research Center for Metabolic Diseases, The Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China.
  • Tang J; Department of Endocrinology and Metabolism, National Clinical Research Center for Metabolic Diseases, The Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China.
  • Zhou CL; Department of Endocrinology and Metabolism, National Clinical Research Center for Metabolic Diseases, The Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China.
  • Tan SW; Department of Endocrinology and Metabolism, National Clinical Research Center for Metabolic Diseases, The Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China.
  • Wang M; Department of Endocrinology, Xiangya Hospital, Central South University, Changsha, Hunan 410008, China.
  • Zhou HD; Department of Endocrinology and Metabolism, National Clinical Research Center for Metabolic Diseases, The Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China.
Mol Cells ; 40(2): 123-132, 2017 Feb.
Article em En | MEDLINE | ID: mdl-28190325

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: MicroRNAs / Proteínas Substratos do Receptor de Insulina Limite: Animals Idioma: En Revista: Mol Cells Assunto da revista: BIOLOGIA MOLECULAR Ano de publicação: 2017 Tipo de documento: Article País de afiliação: China País de publicação:

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: MicroRNAs / Proteínas Substratos do Receptor de Insulina Limite: Animals Idioma: En Revista: Mol Cells Assunto da revista: BIOLOGIA MOLECULAR Ano de publicação: 2017 Tipo de documento: Article País de afiliação: China País de publicação: