Your browser doesn't support javascript.
loading
Chemokine CCL2-CCR2 Signaling Induces Neuronal Cell Death via STAT3 Activation and IL-1ß Production after Status Epilepticus.
Tian, Dai-Shi; Peng, Jiyun; Murugan, Madhuvika; Feng, Li-Jie; Liu, Jun-Li; Eyo, Ukpong B; Zhou, Li-Jun; Mogilevsky, Rochelle; Wang, Wei; Wu, Long-Jun.
Afiliação
  • Tian DS; Department of Neurology, Tongji Hospital, and.
  • Peng J; Department of Cell Biology and Neuroscience, School of Arts and Sciences, Rutgers University, Piscataway, New Jersey 08854, and.
  • Murugan M; Department of Cell Biology and Neuroscience, School of Arts and Sciences, Rutgers University, Piscataway, New Jersey 08854, and.
  • Feng LJ; Department of Neurology, Mayo Clinic, Rochester, Minnesota 55905.
  • Liu JL; Department of Cell Biology and Neuroscience, School of Arts and Sciences, Rutgers University, Piscataway, New Jersey 08854, and.
  • Eyo UB; Department of Neurology, Mayo Clinic, Rochester, Minnesota 55905.
  • Zhou LJ; Department of Cell Biology and Neuroscience, School of Arts and Sciences, Rutgers University, Piscataway, New Jersey 08854, and.
  • Mogilevsky R; Cancer Center, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430022, China.
  • Wang W; Department of Cell Biology and Neuroscience, School of Arts and Sciences, Rutgers University, Piscataway, New Jersey 08854, and.
  • Wu LJ; Department of Cell Biology and Neuroscience, School of Arts and Sciences, Rutgers University, Piscataway, New Jersey 08854, and.
J Neurosci ; 37(33): 7878-7892, 2017 08 16.
Article em En | MEDLINE | ID: mdl-28716963

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Estado Epiléptico / Quimiocina CCL2 / Fator de Transcrição STAT3 / Interleucina-1beta / Receptores CCR2 / Neurônios Limite: Animals Idioma: En Revista: J Neurosci Ano de publicação: 2017 Tipo de documento: Article País de publicação: Estados Unidos

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Estado Epiléptico / Quimiocina CCL2 / Fator de Transcrição STAT3 / Interleucina-1beta / Receptores CCR2 / Neurônios Limite: Animals Idioma: En Revista: J Neurosci Ano de publicação: 2017 Tipo de documento: Article País de publicação: Estados Unidos