Your browser doesn't support javascript.
loading
A ubiquitin-dependent signalling axis specific for ALKBH-mediated DNA dealkylation repair.
Brickner, Joshua R; Soll, Jennifer M; Lombardi, Patrick M; Vågbø, Cathrine B; Mudge, Miranda C; Oyeniran, Clement; Rabe, Renana; Jackson, Jessica; Sullender, Meagan E; Blazosky, Elyse; Byrum, Andrea K; Zhao, Yu; Corbett, Mark A; Gécz, Jozef; Field, Michael; Vindigni, Alessandro; Slupphaug, Geir; Wolberger, Cynthia; Mosammaparast, Nima.
Afiliação
  • Brickner JR; Department of Pathology and Immunology, Division of Laboratory and Genomic Medicine, Washington University School of Medicine, St. Louis, Missouri 63110, USA.
  • Soll JM; Department of Pathology and Immunology, Division of Laboratory and Genomic Medicine, Washington University School of Medicine, St. Louis, Missouri 63110, USA.
  • Lombardi PM; Department of Biophysics and Biophysical Chemistry, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205, USA.
  • Vågbø CB; Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, NTNU, NO-7491 Trondheim, Norway.
  • Mudge MC; PROMEC Core Facility for Proteomics and Metabolomics, NTNU and the Central Norway Regional Health Authority, NO-7491 Trondheim, Norway.
  • Oyeniran C; Department of Pathology and Immunology, Division of Laboratory and Genomic Medicine, Washington University School of Medicine, St. Louis, Missouri 63110, USA.
  • Rabe R; Department of Pathology and Immunology, Division of Laboratory and Genomic Medicine, Washington University School of Medicine, St. Louis, Missouri 63110, USA.
  • Jackson J; Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, NTNU, NO-7491 Trondheim, Norway.
  • Sullender ME; Edward A. Doisy Department of Biochemistry and Molecular Biology, St. Louis University School of Medicine, St. Louis, Missouri 63104, USA.
  • Blazosky E; Department of Pathology and Immunology, Division of Laboratory and Genomic Medicine, Washington University School of Medicine, St. Louis, Missouri 63110, USA.
  • Byrum AK; Department of Biophysics and Biophysical Chemistry, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205, USA.
  • Zhao Y; Department of Pathology and Immunology, Division of Laboratory and Genomic Medicine, Washington University School of Medicine, St. Louis, Missouri 63110, USA.
  • Corbett MA; Department of Pathology and Immunology, Division of Laboratory and Genomic Medicine, Washington University School of Medicine, St. Louis, Missouri 63110, USA.
  • Gécz J; Adelaide Medical School and Robinson Research Institute, The University of Adelaide, Adelaide, South Australia 5000, Australia.
  • Field M; Adelaide Medical School and Robinson Research Institute, The University of Adelaide, Adelaide, South Australia 5000, Australia.
  • Vindigni A; Healthy Mothers and Babies, South Australian Medical Research Institute, Adelaide, South Australia 5000, Australia.
  • Slupphaug G; Genetics of Learning Disability Service, Hunter Genetics, Waratah, New South Wales 2298, Australia.
  • Wolberger C; Edward A. Doisy Department of Biochemistry and Molecular Biology, St. Louis University School of Medicine, St. Louis, Missouri 63104, USA.
  • Mosammaparast N; Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, NTNU, NO-7491 Trondheim, Norway.
Nature ; 551(7680): 389-393, 2017 11 16.
Article em En | MEDLINE | ID: mdl-29144457
Assuntos
Texto completo
Adicionar na Minha BVS
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Transdução de Sinais / Adutos de DNA / Ubiquitina / Complexos Multiproteicos / Reparo do DNA / Síndromes de Tricotiodistrofia / Enzimas AlkB Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Revista: Nature Ano de publicação: 2017 Tipo de documento: Article País de afiliação: Estados Unidos
Texto completo
Adicionar na Minha BVS
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Transdução de Sinais / Adutos de DNA / Ubiquitina / Complexos Multiproteicos / Reparo do DNA / Síndromes de Tricotiodistrofia / Enzimas AlkB Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Revista: Nature Ano de publicação: 2017 Tipo de documento: Article País de afiliação: Estados Unidos