P-Rex1 is dispensable for Erk activation and mitogenesis in breast cancer.

Barrio-Real, Laura; Lopez-Haber, Cynthia; Casado-Medrano, Victoria; Goglia, Alexander G; Toettcher, Jared E; Caloca, Maria J; Kazanietz, Marcelo G

Barrio-Real, Laura; Lopez-Haber, Cynthia; Casado-Medrano, Victoria; Goglia, Alexander G; Toettcher, Jared E; Caloca, Maria J; Kazanietz, Marcelo G.

Afiliação

Barrio-Real L; Department of Systems Pharmacology and Translational Therapeutics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA 19104-6160, USA.
Lopez-Haber C; Department of Systems Pharmacology and Translational Therapeutics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA 19104-6160, USA.
Casado-Medrano V; Department of Systems Pharmacology and Translational Therapeutics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA 19104-6160, USA.
Goglia AG; Department of Molecular Biology, Princeton University, Princeton, NJ 08544-1014, USA.
Toettcher JE; Department of Molecular Biology, Princeton University, Princeton, NJ 08544-1014, USA.
Caloca MJ; Instituto de Biología y Genética Molecular, Consejo Superior de Investigaciones Científicas, Universidad de Valladolid, 47003 Valladolid, Spain.
Kazanietz MG; Department of Systems Pharmacology and Translational Therapeutics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA 19104-6160, USA.

Oncotarget ; 9(47): 28612-28624, 2018 Jun 19.

Article em En | MEDLINE | ID: mdl-29983884

Palavras-chave

Akt; Erk; P-Rex1; Rac1; mitogenesis

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Tipo de estudo: Prognostic_studies Idioma: En Revista: Oncotarget Ano de publicação: 2018 Tipo de documento: Article País de afiliação: Estados Unidos País de publicação: Estados Unidos

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