S-adenosylhomocysteine hydrolase over-expression does not alter S-adenosylmethionine or S-adenosylhomocysteine levels in CBS deficient mice.

Lee, Hyung-Ok; Wang, Liqun; Kuo, Yin-Ming; Andrews, Andrew J; Gupta, Sapna; Kruger, Warren D

Lee, Hyung-Ok; Wang, Liqun; Kuo, Yin-Ming; Andrews, Andrew J; Gupta, Sapna; Kruger, Warren D.

Afiliação

Lee HO; Cancer Biology Program, Fox Chase Cancer Center, Philadelphia, PA, USA.
Wang L; Cancer Biology Program, Fox Chase Cancer Center, Philadelphia, PA, USA.
Kuo YM; Cancer Epigenetics Program, Fox Chase Cancer Center, Philadelphia, PA, USA.
Andrews AJ; Cancer Epigenetics Program, Fox Chase Cancer Center, Philadelphia, PA, USA.
Gupta S; Cancer Biology Program, Fox Chase Cancer Center, Philadelphia, PA, USA.
Kruger WD; Cancer Biology Program, Fox Chase Cancer Center, Philadelphia, PA, USA.

Mol Genet Metab Rep ; 15: 15-21, 2018 Jun.

Article em En | MEDLINE | ID: mdl-30023284

Palavras-chave

AHCY, S-adenosylhomocysteine hydrolase; CBS, cystathionine beta synthase; CMC, carboxymethylcellulose; Cbs−, CBS knockout allele; HA, hemagglutinin; HHcy, hyperhomocysteinemia; Hcy, homocysteine; Met, methionine; Metabolism; Methionine; SAH, S-adenosyl homocysteine; SAM, S-adenosyl methionine; Tg-I278T, transgene human CBS containing the I278T mutation; Transgenic; Zn, zinc water; tHcy, total homocysteine

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Idioma: En Revista: Mol Genet Metab Rep Ano de publicação: 2018 Tipo de documento: Article País de afiliação: Estados Unidos País de publicação: Estados Unidos

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