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Loss of the mitochondrial i-AAA protease YME1L leads to ocular dysfunction and spinal axonopathy.
Sprenger, Hans-Georg; Wani, Gulzar; Hesseling, Annika; König, Tim; Patron, Maria; MacVicar, Thomas; Ahola, Sofia; Wai, Timothy; Barth, Esther; Rugarli, Elena I; Bergami, Matteo; Langer, Thomas.
Afiliação
  • Sprenger HG; Max-Planck-Institute for Biology of Ageing, Cologne, Germany.
  • Wani G; Institute of Genetics and Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany.
  • Hesseling A; Institute of Genetics and Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany.
  • König T; Institute of Genetics and Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany.
  • Patron M; Institute of Genetics and Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany.
  • MacVicar T; Max-Planck-Institute for Biology of Ageing, Cologne, Germany.
  • Ahola S; Institute of Genetics and Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany.
  • Wai T; Max-Planck-Institute for Biology of Ageing, Cologne, Germany.
  • Barth E; Institute of Genetics and Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany.
  • Rugarli EI; Max-Planck-Institute for Biology of Ageing, Cologne, Germany.
  • Bergami M; Institute of Genetics and Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany.
  • Langer T; Institute of Genetics and Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany.
EMBO Mol Med ; 11(1)2019 01.
Article em En | MEDLINE | ID: mdl-30389680
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Metaloendopeptidases / Doenças Mitocondriais / ATPases Associadas a Diversas Atividades Celulares / Doenças do Sistema Nervoso Tipo de estudo: Etiology_studies Limite: Animals Idioma: En Revista: EMBO Mol Med Assunto da revista: BIOLOGIA MOLECULAR Ano de publicação: 2019 Tipo de documento: Article País de afiliação: Alemanha País de publicação: Reino Unido
Texto completo
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Metaloendopeptidases / Doenças Mitocondriais / ATPases Associadas a Diversas Atividades Celulares / Doenças do Sistema Nervoso Tipo de estudo: Etiology_studies Limite: Animals Idioma: En Revista: EMBO Mol Med Assunto da revista: BIOLOGIA MOLECULAR Ano de publicação: 2019 Tipo de documento: Article País de afiliação: Alemanha País de publicação: Reino Unido