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Differential induction of interferon stimulated genes between type I and type III interferons is independent of interferon receptor abundance.
Pervolaraki, Kalliopi; Rastgou Talemi, Soheil; Albrecht, Dorothee; Bormann, Felix; Bamford, Connor; Mendoza, Juan L; Garcia, K Christopher; McLauchlan, John; Höfer, Thomas; Stanifer, Megan L; Boulant, Steeve.
Afiliação
  • Pervolaraki K; Schaller research group at CellNetworks, Department of Infectious Diseases, Virology, Heidelberg University Hospital, Heidelberg, Germany.
  • Rastgou Talemi S; Division of Cellular polarity and viral infection, German Cancer Research Center (DKFZ), Heidelberg, Germany.
  • Albrecht D; Division of Theoretical Systems Biology, German Cancer Research Center (DKFZ), Heidelberg, Germany.
  • Bormann F; BioQuant Center, Heidelberg University, Heidelberg, Germany.
  • Bamford C; Schaller research group at CellNetworks, Department of Infectious Diseases, Virology, Heidelberg University Hospital, Heidelberg, Germany.
  • Mendoza JL; Division of Epigenetics, German Cancer Research Center (DKFZ), Heidelberg, Germany.
  • Garcia KC; MRC- University of Glasgow Centre for Virus Research, Glasgow, United Kingdom.
  • McLauchlan J; Howard Hughes Medical Institute, Department of Molecular and Cellular Physiology and Department of Structural Biology, Stanford University School of Medicine, Stanford, CA, United States of America.
  • Höfer T; Howard Hughes Medical Institute, Department of Molecular and Cellular Physiology and Department of Structural Biology, Stanford University School of Medicine, Stanford, CA, United States of America.
  • Stanifer ML; MRC- University of Glasgow Centre for Virus Research, Glasgow, United Kingdom.
  • Boulant S; Division of Theoretical Systems Biology, German Cancer Research Center (DKFZ), Heidelberg, Germany.
PLoS Pathog ; 14(11): e1007420, 2018 11.
Article em En | MEDLINE | ID: mdl-30485383
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Interferon Tipo I / Interferons Limite: Humans Idioma: En Revista: PLoS Pathog Ano de publicação: 2018 Tipo de documento: Article País de afiliação: Alemanha País de publicação: Estados Unidos
Texto completo
Adicionar na Minha BVS
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XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Interferon Tipo I / Interferons Limite: Humans Idioma: En Revista: PLoS Pathog Ano de publicação: 2018 Tipo de documento: Article País de afiliação: Alemanha País de publicação: Estados Unidos