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IFNγ induces epigenetic programming of human T-bethi B cells and promotes TLR7/8 and IL-21 induced differentiation.
Zumaquero, Esther; Stone, Sara L; Scharer, Christopher D; Jenks, Scott A; Nellore, Anoma; Mousseau, Betty; Rosal-Vela, Antonio; Botta, Davide; Bradley, John E; Wojciechowski, Wojciech; Ptacek, Travis; Danila, Maria I; Edberg, Jeffrey C; Bridges, S Louis; Kimberly, Robert P; Chatham, W Winn; Schoeb, Trenton R; Rosenberg, Alexander F; Boss, Jeremy M; Sanz, Ignacio; Lund, Frances E.
Afiliação
  • Zumaquero E; Department of Microbiology, The University of Alabama at Birmingham, Birmingham, United States.
  • Stone SL; Department of Microbiology, The University of Alabama at Birmingham, Birmingham, United States.
  • Scharer CD; Department of Microbiology and Immunology, Division of Rheumatology, Emory University, Atlanta, United States.
  • Jenks SA; Department of Medicine, Division of Rheumatology, Emory University, Atlanta, United States.
  • Nellore A; Department of Medicine, Division of Infectious Disease, The University of Alabama at Birmingham, Birmingham, United States.
  • Mousseau B; Department of Microbiology, The University of Alabama at Birmingham, Birmingham, United States.
  • Rosal-Vela A; Department of Microbiology, The University of Alabama at Birmingham, Birmingham, United States.
  • Botta D; Department of Microbiology, The University of Alabama at Birmingham, Birmingham, United States.
  • Bradley JE; Department of Medicine, Division of Clinical Immunology and Rheumatology, The University of Alabama at Birmingham, Birmingham, United States.
  • Wojciechowski W; Center for Pediatric Biomedical Research, Flow Cytometry Shared Resource Laboratory, University of Rochester School of Medicine and Dentistry, Rochester, United States.
  • Ptacek T; Department of Microbiology, The University of Alabama at Birmingham, Birmingham, United States.
  • Danila MI; Informatics Group, Center for Clinical and Translational Science, The University of Alabama at Birmingham, Birmingham, United States.
  • Edberg JC; Department of Medicine, Division of Clinical Immunology and Rheumatology, The University of Alabama at Birmingham, Birmingham, United States.
  • Bridges SL; Department of Medicine, Division of Clinical Immunology and Rheumatology, The University of Alabama at Birmingham, Birmingham, United States.
  • Kimberly RP; Department of Medicine, Division of Clinical Immunology and Rheumatology, The University of Alabama at Birmingham, Birmingham, United States.
  • Chatham WW; Department of Medicine, Division of Clinical Immunology and Rheumatology, The University of Alabama at Birmingham, Birmingham, United States.
  • Schoeb TR; Department of Medicine, Division of Clinical Immunology and Rheumatology, The University of Alabama at Birmingham, Birmingham, United States.
  • Rosenberg AF; Department of Genetics, Animal Resources Program, The University of Alabama at Birmingham, Birmingham, United States.
  • Boss JM; Department of Microbiology, The University of Alabama at Birmingham, Birmingham, United States.
  • Sanz I; The Informatics Institute, The University of Alabama at Birmingham, Birmingham, United States.
  • Lund FE; Department of Microbiology and Immunology, Division of Rheumatology, Emory University, Atlanta, United States.
Elife ; 82019 05 15.
Article em En | MEDLINE | ID: mdl-31090539
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Diferenciação Celular / Subpopulações de Linfócitos B / Interleucinas / Interferon gama / Epigênese Genética / Receptor 7 Toll-Like / Receptor 8 Toll-Like Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Revista: Elife Ano de publicação: 2019 Tipo de documento: Article País de afiliação: Estados Unidos País de publicação: Reino Unido
Texto completo
Adicionar na Minha BVS
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Diferenciação Celular / Subpopulações de Linfócitos B / Interleucinas / Interferon gama / Epigênese Genética / Receptor 7 Toll-Like / Receptor 8 Toll-Like Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Revista: Elife Ano de publicação: 2019 Tipo de documento: Article País de afiliação: Estados Unidos País de publicação: Reino Unido