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M1-like macrophage-derived exosomes suppress angiogenesis and exacerbate cardiac dysfunction in a myocardial infarction microenvironment.
Liu, Shaojun; Chen, Jing; Shi, Jian; Zhou, Wenyi; Wang, Li; Fang, Weilun; Zhong, Yun; Chen, Xiaohui; Chen, Yanfang; Sabri, Abdelkarim; Liu, Shiming.
Afiliação
  • Liu S; Guangzhou Institute of Cardiovascular Disease, Guangdong Key Laboratory of Vascular Diseases, State Key Laboratory of Respiratory Disease, The Second Affiliated Hospital, Guangzhou Medical University, Guangzhou, 510260, Guangdong, People's Republic of China. shaojunliu@gzhmu.edu.cn.
  • Chen J; Guangzhou Institute of Cardiovascular Disease, Guangdong Key Laboratory of Vascular Diseases, State Key Laboratory of Respiratory Disease, The Second Affiliated Hospital, Guangzhou Medical University, Guangzhou, 510260, Guangdong, People's Republic of China.
  • Shi J; Guangzhou Institute of Cardiovascular Disease, Guangdong Key Laboratory of Vascular Diseases, State Key Laboratory of Respiratory Disease, The Second Affiliated Hospital, Guangzhou Medical University, Guangzhou, 510260, Guangdong, People's Republic of China.
  • Zhou W; Guangzhou Institute of Cardiovascular Disease, Guangdong Key Laboratory of Vascular Diseases, State Key Laboratory of Respiratory Disease, The Second Affiliated Hospital, Guangzhou Medical University, Guangzhou, 510260, Guangdong, People's Republic of China.
  • Wang L; Guangzhou Institute of Cardiovascular Disease, Guangdong Key Laboratory of Vascular Diseases, State Key Laboratory of Respiratory Disease, The Second Affiliated Hospital, Guangzhou Medical University, Guangzhou, 510260, Guangdong, People's Republic of China.
  • Fang W; Department of Cardiology, The Second Affiliated Hospital of Guangzhou Medical University, Guangzhou Medical University, Guangzhou, 510260, Guangdong, People's Republic of China.
  • Zhong Y; Guangzhou Institute of Cardiovascular Disease, Guangdong Key Laboratory of Vascular Diseases, State Key Laboratory of Respiratory Disease, The Second Affiliated Hospital, Guangzhou Medical University, Guangzhou, 510260, Guangdong, People's Republic of China.
  • Chen X; Guangzhou Institute of Cardiovascular Disease, Guangdong Key Laboratory of Vascular Diseases, State Key Laboratory of Respiratory Disease, The Second Affiliated Hospital, Guangzhou Medical University, Guangzhou, 510260, Guangdong, People's Republic of China.
  • Chen Y; Department of Cardiology, The Second Affiliated Hospital of Guangzhou Medical University, Guangzhou Medical University, Guangzhou, 510260, Guangdong, People's Republic of China.
  • Sabri A; Department of Emergency, The Second Affiliated Hospital of Guangzhou Medical University, Guangzhou Medical University, Guangzhou, 510260, Guangdong, People's Republic of China.
  • Liu S; Guangzhou Institute of Cardiovascular Disease, Guangdong Key Laboratory of Vascular Diseases, State Key Laboratory of Respiratory Disease, The Second Affiliated Hospital, Guangzhou Medical University, Guangzhou, 510260, Guangdong, People's Republic of China.
Basic Res Cardiol ; 115(2): 22, 2020 02 28.
Article em En | MEDLINE | ID: mdl-32112145
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Função Ventricular Esquerda / Neovascularização Fisiológica / Células Endoteliais / Exossomos / Microambiente Celular / Macrófagos / Infarto do Miocárdio / Miocárdio Limite: Animals / Humans / Male Idioma: En Revista: Basic Res Cardiol Ano de publicação: 2020 Tipo de documento: Article País de publicação: Alemanha
Texto completo
Adicionar na Minha BVS
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Função Ventricular Esquerda / Neovascularização Fisiológica / Células Endoteliais / Exossomos / Microambiente Celular / Macrófagos / Infarto do Miocárdio / Miocárdio Limite: Animals / Humans / Male Idioma: En Revista: Basic Res Cardiol Ano de publicação: 2020 Tipo de documento: Article País de publicação: Alemanha