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Gap junctions amplify TRPV4 activation-initiated cell injury via modification of intracellular Ca2+ and Ca2+-dependent regulation of TXNIP.
Zhang, Xiling; Mao, Zhimin; Huang, Yanru; Zhang, Zhen; Yao, Jian.
Afiliação
  • Zhang X; Department of Urology, The Fourth Affiliated Hospital of China Medical University , Shenyang, China.
  • Mao Z; Division of Molecular Signaling, Department of the Advanced Biomedical Research, Interdisciplinary Graduate School of Medicine, University of Yamanashi , Chuo, Japan.
  • Huang Y; Division of Molecular Signaling, Department of the Advanced Biomedical Research, Interdisciplinary Graduate School of Medicine, University of Yamanashi , Chuo, Japan.
  • Zhang Z; Division of Molecular Signaling, Department of the Advanced Biomedical Research, Interdisciplinary Graduate School of Medicine, University of Yamanashi , Chuo, Japan.
  • Yao J; Division of Molecular Signaling, Department of the Advanced Biomedical Research, Interdisciplinary Graduate School of Medicine, University of Yamanashi , Chuo, Japan.
Channels (Austin) ; 14(1): 246-256, 2020 12.
Article em En | MEDLINE | ID: mdl-32752916

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Cálcio / Junções Comunicantes / Proteínas de Ciclo Celular / Canais de Cátion TRPV Limite: Animals Idioma: En Revista: Channels (Austin) Ano de publicação: 2020 Tipo de documento: Article País de afiliação: China País de publicação: Estados Unidos

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Cálcio / Junções Comunicantes / Proteínas de Ciclo Celular / Canais de Cátion TRPV Limite: Animals Idioma: En Revista: Channels (Austin) Ano de publicação: 2020 Tipo de documento: Article País de afiliação: China País de publicação: Estados Unidos