[Progress on the effects of cancer-related fibroblast induced by Helicobacter pylori infection on gastric epithelial-mesenchymal transition].
Sheng Li Xue Bao
; 76(4): 547-560, 2024 Aug 25.
Article
em Zh
| MEDLINE
| ID: mdl-39192788
ABSTRACT
Helicobacter pylori (Hp) is a Gram-negative bacterium that colonizes in the gastric mucosa. Hp induces the production of cancer-associated fibroblasts (CAF) in the stomach. The virulence factors of Hp and CAF trigger epithelial-mesenchymal transition (EMT), leading to local inflammation, damage to the gastric mucosa, and the occurrence of chronic gastritis. Here, we summarize the molecular mechanisms of CAF mediated gastric EMT after Hp infection, providing new insights into potential molecular targets and strategies for the future treatment of Hp infection associated gastric cancer.
Buscar no Google
Coleções:
01-internacional
Base de dados:
MEDLINE
Assunto principal:
Neoplasias Gástricas
/
Helicobacter pylori
/
Infecções por Helicobacter
/
Transição Epitelial-Mesenquimal
/
Mucosa Gástrica
Limite:
Animals
/
Humans
Idioma:
Zh
Revista:
Sheng Li Xue Bao
Ano de publicação:
2024
Tipo de documento:
Article
País de afiliação:
China
País de publicação:
China