A single session of aerobic exercise reduces systolic blood pressure at rest and in response to stress in women with rheumatoid arthritis and hypertension.

Rheumatoid arthritis (RA) is an autoimmune inflammatory disease characterized by increased risk of cardiovascular disease and hypertension (HT). A single session of aerobic exercise may reduce blood pressure (BP) in different clinical groups; however, little is known about the acute effects of exercise on BP in RA patients. This is a randomized controlled crossover study that assessed the effects of a single session of aerobic exercise on resting BP, on BP responses to stressful stimuli, and on 24-h BP in women with RA and HT. Twenty women with RA and HT (53 ± 10 years) undertook sessions of 30-min treadmill exercise (50% VO2max) or control (no exercise) in a crossover fashion. Before and after the sessions, BP was measured at rest, and in response to the Stroop-Color Word Test (SCWT), the Cold Pressor Test (CPT), and an isometric handgrip test. After the sessions, participants were also fitted with an ambulatory BP monitor for the assessment of 24-h BP. A single session of exercise reduced resting systolic BP (SBP) (-5 ± 9 mmHg; p < 0.05), and reduced SBP response to the SCWT (-7 ± 14 mmHg; p < 0.05), and to the CPT (-5 ± 11 mmHg; p < 0.05). Exercise did not reduce resting diastolic BP (DBP), BP responses to the isometric handgrip test or 24-h BP. In conclusion, a single session of aerobic exercise reduced SBP at rest and in response to stressful stimuli in hypertensive women with RA. These results support the use of exercise as a strategy for controlling HT and, hence, reducing cardiovascular risk in women with RA.Clinical Trial Registration: This study registered at the Brazilian Clinical Trials ( https://ensaiosclinicos.gov.br/rg/RBR-867k9g ) at 12/13/2019.

Post-dynamic, isometric and combined resistance exercise responses in medicated hypertensive men.

This study investigated the effects of dynamic resistance exercise (DRE), isometric handgrip exercise (IHE) and combined resistance exercise (DRE+IHE) on post-exercise hypotension (PEH) and its hemodynamic, autonomic, and vascular mechanisms. For that, 70 medicated hypertensives men (52 ± 8 years) were randomly allocated to perform one of the following interventions: DRE (3 sets, 8 exercises, 50% of 1RM), IHE (4 sets, 2 min, 30% of MVC), CRE (DRE+IHE) and control (CON, seated rest). Before and after the interventions, blood pressure (BP), systemic hemodynamics, cardiovascular autonomic modulation and brachial vascular parameters were evaluated. After the DRE and CRE, systolic and mean BP decreased (SBP = -7 ± 6 and -8 ± 8 mmHg; MBP -4 ± 5 and -5 ± 5 mmHg, respectively, all P < 0.05), vascular conductance increased (+ 0.47 ± 0.61 and +0.40 ± 0.47 ml.min-1.mmHg-1, respectively, both P < 0.05) and baroreflex sensitivity decreased (-0.15 ± 0.38 and -0.29 ± 0.47 ms/mmHg, respectively, both P < 0.05) in comparison to pre-exercise values. No variable presented any significant change after IHE. The responses observed after CRE were similar to DRE and significantly different from CON and IHE. In conclusion, DRE, but not IHE, elicits PEH, which happens concomitantly to skeletal muscle vasodilation and decreased baroreflex sensitivity. Moreover, adding IHE to DRE does not potentiate PEH and neither changes its mechanisms.Clinical Trial Registration: Data from this study derived from an ongoing longitudinal clinical trial approved by the Institution's Ethics Committee of Human Research (process 2.870.688) and registered at the Brazilian Clinical Trials (RBR-4fgknb) at http://www.ensaiosclinicos.gov.br .

Can blood pressure decrease after maximal exercise test predict the blood pressure lowering effect of aerobic training in treated hypertensive men?

The acute decrease in blood pressure (BP) observed after a session of exercise (called post-exercise hypotension) has been proposed as a tool to predict the chronic reduction in BP induced by aerobic training. Therefore, this study investigated whether post-exercise hypotension observed after a maximal exercise test is associated to the BP-lowering effect of aerobic training in treated hypertensives. Thirty hypertensive men (50 ± 8 years) who were under consistent anti-hypertensive treatment underwent a maximal exercise test (15 watts/min until exhaustion), and post-exercise hypotension was determined by the difference between BP measured before and at 30 min after the test. Subsequently, the patients underwent 10 weeks of aerobic training (3 times/week, 45 min/session at moderate intensity), and the BP-lowering effect of training was assessed by the difference in BP measured before and after the training period. Pearson correlations were employed to evaluate the associations. Post-maximal exercise test hypotension was observed for systolic and mean BPs (-8 ± 6 and -2 ± 4 mmHg, all P < 0.05). Aerobic training reduced clinic systolic/diastolic BPs (-5 ± 6/-2 ± 3 mmHg, both P < 0.05) as well as awake and 24 h mean BPs (-2 ± 6 and -2 ± 5 mmHg, all P < 0.05). No significant correlation was detected between post-exercise hypotension and the BP-lowering effect of training either for clinic or ambulatory BPs (r values ranging from 0.00 to 0.32, all p > 0.05). Post-exercise hypotension assessed 30 min after a maximal exercise test cannot be used to predict the BP-lowering effect of aerobic training in treated hypertensive men.

Consistency of hemodynamic and autonomic mechanisms underlying post-exercise hypotension.

Post-exercise hypotension (PEH) is a clinically relevant phenomenon, but its mechanisms vary between different studies and between the participants within each study. Additionally, it is possible that PEH mechanisms are not consistent in each individual (i.e. within-individual variation), which has not been investigated yet. Thus, the aim of the current study was to assess the within-individual consistency of PEH hemodynamic and autonomic mechanisms. For that, 30 subjects performed 4 sessions divided in 2 blocks (test and retest). In each block, an exercise (cycling, 45 min, 50%VO2peak) and a control (seated rest, 45 min) session was randomly conducted. Blood pressure (BP) and its mechanisms were evaluated pre- and post-interventions. In each block, individual responses were calculated as post-exercise minus post-control, and a response was considered present when its magnitude reached the typical error of the measurement. Consistencies were evaluated by comparing test and retest responses through kappa coefficient (k). PEH consistency was calculated using role sample, while mechanisms consistency was evaluated in those with consistent PEH. Twenty-one (70%) participants showed consistent PEH, 5 (17%) presented PEH in only test or retest and 4 (13%) had absent PEH response, characterising a good consistency (k = 0.510). Regarding mechanisms' responses, good consistency was found for heart rate (k = 0.456), sympathovagal balance (k = 0.438), and baroreflex sensitivity (k = 0.458); while systemic vascular resistance (k = 0.152), cardiac output (k = -0.400), stroke volume (k = -0.055), and sympathetic vasomotor modulation (k = -0.096) presented marginal consistencies. Thus, PEH is a highly consistent physiological phenomenon, although its mechanisms present variable consistencies.

Activation of Mechanoreflex, but not Central Command, Delays Heart Rate Recovery after Exercise in Healthy Men.

This study tested the hypotheses that activation of central command and muscle mechanoreflex during post-exercise recovery delays fast-phase heart rate recovery with little influence on the slow phase. Twenty-five healthy men underwent three submaximal cycling bouts, each followed by a different 5-min recovery protocol: active (cycling generated by the own subject), passive (cycling generated by external force) and inactive (no-cycling). Heart rate recovery was assessed by the heart rate decay from peak exercise to 30 s and 60 s of recovery (HRR30s, HRR60s fast phase) and from 60 s-to-300 s of recovery (HRR60-300s slow phase). The effect of central command was examined by comparing active and passive recoveries (with and without central command activation) and the effect of mechanoreflex was assessed by comparing passive and inactive recoveries (with and without mechanoreflex activation). Heart rate recovery was similar between active and passive recoveries, regardless of the phase. Heart rate recovery was slower in the passive than inactive recovery in the fast phase (HRR60s=20±8vs.27 ±10 bpm, p<0.01), but not in the slow phase (HRR60-300s=13±8vs.10±8 bpm, p=0.11). In conclusion, activation of mechanoreflex, but not central command, during recovery delays fast-phase heart rate recovery. These results elucidate important neural mechanisms behind heart rate recovery regulation.

Effects of ACEi and ARB on post-exercise hypotension induced by exercises conducted at different times of day in hypertensive men.

BACKGROUND: Post-exercise hypotension (PEH) is greater after evening than morning exercise, but antihypertensive drugs may affect the evening potentiation of PEH. Objective: To compare morning and evening PEH in hypertensives receiving angiotensin-converting enzyme inhibitors (ACEi) or angiotensin II receptor blockers (ARB). METHODS: Hypertensive men receiving ACEi (n = 14) or ARB (n = 15) underwent, in a random order, two maximal exercise tests (cycle ergometer, 15 watts/min until exhaustion) with one conducted in the morning (7 and 9 a.m.) and the other in the evening (8 and 10 p.m.). Auscultatory blood pressure (BP) was assessed in triplicate before and 30 min after the exercises. Changes in BP (post-exercise - pre-exercise) were compared between the groups and the sessions using a two-way mixed ANOVA and considering P < .05 as significant. RESULTS: In the ARB group, systolic BP decrease was greater after the evening than the morning exercise, while in the ACEi group, it was not different after the exercises conducted at the different times of the day. Additionally, after the evening exercise, systolic BP decrease was lower in the ACEi than the ARB group (ARB = -11 ± 8 vs -6 ± 6 and ACEi = -6 ± 7 vs. -8 ± 5 mmHg, evening vs. morning, respectively, P for interaction = 0.014). CONCLUSIONS: ACEi, but not ARB use, blunts the greater PEH that occurs after exercise conducted in the evening than in the morning.

Post-exercise hypotension and its hemodynamic determinants depend on the calculation approach.

Post-exercise hypotension (PEH) has been assessed by three calculation approaches: I = (post-exercise - pre-exercise), II = (post-exercise - post-control), and III = [(post-exercise - pre-exercise) - (post-control - pre-control)]. This study checked whether these calculation approaches influence PEH and its determinants. For that, 30 subjects underwent two exercise (cycling, 45 min, 50% VO2 peak) and two control (seated rest, 45 min) sessions. Systolic (SBP) and diastolic (DBP) blood pressures, cardiac output (CO), systemic vascular resistance (SVR), heart rate (HR), and stroke volume (SV) were measured pre- and post-interventions in each session. The mean value for each moment in each type of session was calculated, and responses to exercise were analyzed with each approach (I, II, and III) to evaluate the occurrence of PEH and its determinants. Systolic PEH was significant when calculated by all approaches (I = -5 ± 1, II = -11 ± 2, and III = -11 ± 2 mmHg, p < 0.05), while diastolic PEH was only significant when calculated by approaches II and III (-6 ± 1 and -6 ± 1 mmHg, respectively, p < 0.05). CO decreased significantly after the exercise when calculated by approach I, but remained unchanged with approaches II and III, while SVR increased significantly with approach I, but decreased significantly with approaches II and III. HR was unchanged after the exercise with approach I, but increased significantly with approaches II and III, while SV decreased significantly with all approaches. Thus, PEH and its hemodynamic determinants are influenced by the calculation approach, which should be considered when designing, analyzing, and comparing PEH studies.

Effects of postexercise cooling on heart rate recovery in normotensive and hypertensive men.

BACKGROUND: Postexercise heart rate recovery (HRR) is determined by cardiac autonomic restoration after exercise and is reduced in hypertension. Postexercise cooling accelerates HRR in healthy subjects, but its effects in a population with cardiac autonomic dysfunction, such as hypertensives (HT), may be blunted. This study assessed and compared the effects of postexercise cooling on HRR and cardiac autonomic regulation in HT and normotensive (NT) subjects. METHODS: Twenty-three never-treated HT (43 ± 8 years) and 25 NT (45 ± 8 years) men randomly underwent two exercise sessions (30 min of cycling at 70% VO2peak ) followed by 15 min of recovery. In one randomly allocated session, a fan was turned on in front of the subject during the recovery (cooling), while in the other session, no cooling was performed (control). HRR was assessed by heart rate reductions after 60 s (HRR60s) and 300 s (HRR300s) of recovery, short-term time constant of HRR (T30) and the time constant of the HRR after exponential fitting (HRRτ). HRV was assessed using time- and frequency-domain indices. RESULTS: HRR and HRV responses in the cooling and control sessions were similar between the HT and NT. Thus, in both groups, postexercise cooling equally accelerated HRR (HRR300s = 39±12 versus 36 ± 10 bpm, P≤0·05) and increased postexercise HRV (lnRMSSD = 1·8 ± 0·7 versus 1·6 ± 0·7 ms, P≤0·05). CONCLUSION: Differently from the hypothesis, postexercise cooling produced similar improvements in HRR in HT and NT men, likely by an acceleration of cardiac parasympathetic reactivation and sympathetic withdrawal. These results suggest that postexercise cooling equally accelerates HRR in hypertensive and normotensive subjects.

Exercício físico e pressão arterial: efeitos, mecanismos, influências e implicações na hipertensão arterial / Physical exercise and blood pressure: effects, mechanisms, influences and impacts on hypertension

O exercício físico é recomendado no tratamento da hipertensão arterial. Agudamente, a execução do exercício promove aumento da pressão arterial (PA), mas, no período de recuperação pós-exercício, é possível evidenciar redução da PA e, principalmente, após um período de treinamento físico crônico, pode haver diminuição da PA clínica e de 24 horas dos hipertensos. Apesar desses efeitos serem conhecidos, sua magnitude e mecanismos dependem do tipo de exercício executado e de suas características. Este artigo revê os efeitos agudos e crônicos clássicos do exercício aeróbico e os efeitos mais recentemente estudados dos exercícios resistidos isométrico e dinâmico na PA, seus mecanismos e fatores de influência, ressaltando os pontos que embasam as recomendações atuais sobre o uso do exercício na hipertensão arterial. O conhecimento atual demonstra que: 1) o exercício aeróbico promove aumento da PA sistólica durante sua execução, gera hipotensão pós-exercício clinicamente relevante e reduz a PA clínica e de 24 horas após o treinamento; 2) o exercício resistido isométrico promove aumento progressivo da PA sistólica e diastólica durante sua execução, não produz hipotensão pós-exercício consistente e reduz a PA clínica após o treinamento, mas esse efeito hipotensor ocorre com um protocolo específico de exercício de handgrip; e 3) o exercício resistido dinâmico promove grande aumento da PA sistólica e diastólica durante sua execução, gera hipotensão pós-exercício cuja relevância clínica ainda precisa ser comprovada e parece diminuir a PA clínica, mas não a ambulatorial, após o treinamento. Face a esses conhecimentos, o treinamento aeróbico complementado pelo resistido dinâmico é recomendado na hipertensão

Physical exercise is recommended for hypertension treatment. Acutely, exercise execution increases blood pressure (BP), but, during the recovery period, BP decreases, and after a chronic training period, clinic and ambulatory BP may decrease in hypertensives. Despite these known effects of exercise, their magnitude and mechanisms depend on the type of exercise and its characteristics. This article reviews the classical acute and chronic effects of aerobic exercise and the more recent knowledge about isometric and dynamic resistance exercises on BP, its mechanisms and factors of influence, highlighting the aspects underlying exercise recommendations for hypertension. Current scientific knowledge shows that: 1) aerobic exercise increases systolic BP during its execution, produces a clinically significant post-exercise hypotension, and chronically decreases clinic and 24-hour BP; 2) isometric resistance exercise produces a progressive increase in systolic and diastolic BP during its execution, does not promote consistent post-exercise hypotension, and decreases clinic BP after training, but this hypotensive effect results from a specific protocol of isometric handgrip; and 3) dynamic resistance exercise produces a huge progressive increase in systolic and diastolic BP during its execution, promotes post-exercise hypotension with questionable clinical relevance, and seems to decrease clinic but not ambulatory BP after training. Based on this current knowledge, regular aerobic exercise complemented by dynamic resistance exercise is recommended for hypertension

Recommendations in Post-exercise Hypotension: Concerns, Best Practices and Interpretation.

Post-exercise hypotension (PEH) is a clinically relevant phenomenon that has been widely investigated. However, the characteristics of study designs, such as familiarization to blood pressure measurements, duration of PEH assessments or strategies to analyze PEH present discrepancies across studies. Thus identifying key points to standardize across PEH studies is necessary to help researchers to build stronger study designs, to facilitate comparisons across studies, and to avoid misinterpretations of results. The goal of this narrative review of methods used in PEH studies was therefore to gather and find possible influencers in the characteristics of study design and strategies to analyze blood pressure. Data found in this review suggest that PEH studies should have at least two familiarization screening visits, and should assess blood pressure for at least 20 min, but preferably for 120 min, during recovery from exercise. Another important aspect is the strategy to analyze PEH, which may lead to different interpretations. This information should guide a priori study design decisions.

Aerobic Stimulus Induced by Virtual Reality Games in Stroke Survivors.

OBJECTIVE: To evaluate whether virtual reality games (VRGs) in stroke survivors produce significant and reproducible heart rate and oxygen consumption (V˙o2) responses during their execution, corresponding to an intensity between the anaerobic threshold (AT) and the respiratory compensation point (RCP). DESIGN: Single-subject, repeated-measure design. SETTING: Stroke survivors registered from a rehabilitation program. PARTICIPANTS: Chronic hemiparetic stroke survivors (N=12; 10 men; mean age ± SD, 58±12y) rated at 3 or 4 in the Functional Ambulation Categories. INTERVENTIONS: Participants underwent, in a random order, 2 identical sessions of VRGs (console Xbox 360 + Kinect) and 1 control session (38min watching a movie). The VRG sessions were composed of 4 sets of VRGs (3min of tennis, 1min for changing the game, and 4min of boxing) interspaced with 2 minutes of rest. MAIN OUTCOME MEASURES: Heart rate and V˙o2 were measured during the experimental sessions and compared with heart rate and V˙o2 obtained at AT and RCP assessed during a maximal cardiopulmonary exercise test. RESULTS: Heart rate and V˙o2 during VRGs had good reproducibility (intraclass correlation coefficients, ≥.91 and ≥.85, respectively; coefficients of variation, ≤6.7% and ≤13.7%, respectively). Heart rate during VRGs was similar to AT and significantly lower than RCP (P≤.05), while V˙o2 was significantly lower than AT and RCP (P<.05). CONCLUSIONS: An acute session of VRGs composed of tennis and boxing games using the console XBox 360 + Kinect promotes reproducible responses of heart rate and V˙o2 that corresponded, respectively, to AT and below AT, characterizing a low-intensity aerobic stimulus.

Reproducibility of heart rate recovery in patients with intermittent claudication.

BACKGROUND: Postexercise heart rate recovery (HRR) is a non-invasive tool for cardiac autonomic function assessment. Reproducibility of HRR has been established in healthy subjects; however, no study has evaluated this reproducibility in clinical populations who may present autonomic dysfunction. Patients with peripheral artery disease and intermittent claudication (IC) often present altered cardiac autonomic function and HRR could be an interesting tool for evaluating autonomic responses to interventions in this population. Therefore, the reproducibility of HRR should be determined in this specific population. OBJECTIVE: To determine the reproducibility of HRR indices in patients with IC. METHODS: Nineteen men with IC underwent two repeated maximal treadmill tests. Raw HR and relative HRR (difference to exercise peak) indices measured at 30, 60, 120, 180, 240 and 300s of recovery were evaluated. The presence of systematic bias was assessed by comparing test and retest mean values via paired t-test. Reliability was assessed by intraclass correlation coefficient (ICC), and agreement by typical error (TE), coefficient of variation (CV) and minimal detectable difference (MDD). RESULTS: There were no significant differences between the test and retest values of all raw HR and relative HRR indices (P ≥ 0·05), except for HR120s (P = 0·032). All indices exhibited excellent reliability (ICC ≥ 0·78). Raw HR and relative HRR indices showed TEs ≤ 6·4 bpm and MDDs ≤ 17·8 bpm. In addition, all indices showed CVs ≤ 13·2%, except HRR30s (CV = 45·6%). CONCLUSIONS: The current results demonstrated that most HRR indices were highly reproducible with no systematic error, excellent reliability and good agreement in patients with IC following maximal graded exercise.

Reproducibility (Reliability and Agreement) of Post-exercise Hypotension.

This study determined the reproducibility of post-exercise hypotension (PEH) calculated by the following methods: PEH_I=post-exercise BP - pre-exercise BP; PEH_II=post-exercise BP - post-control BP; and PEH_III=[(post-exercise BP - pre-exercise BP)-(post-control BP - pre-control BP)]. Twenty-five participants underwent four sessions divided in two blocks (test and retest). Each block consisted of one exercise and one control session. BP pressure was measured before and after the interventions. The presence of systematic error (paired t-tests), reliability [intraclass coefficient correlation (ICC)], and agreement [typical error (TE) and minimal detectable difference (MDD)] were evaluated. PEHs calculated by the three methods were similar between test and retest. For systolic PEH, ICC was>0.74, TE ranged from 2.6 to 4.6 mmHg and MDD from 7.2 to 12.8 mmHg for the three methods. For diastolic PEH, ICC was<0.48, TE ranged from 3.5 to 5.6 mmHg and MDD from 9.8 to 15.4 mmHg for the three methods. Thus, systolic PEH calculated by the three methods has good/excellent reliability, while diastolic PEH has fair/poor reliability. Regarding agreement, TE and MDD varied among the methods, which implies that the specific parameters given for each method should be used to estimate sample sizes for studies and the minimal individual difference considered real when comparing PEHs.

Metaboreflex activation delays heart rate recovery after aerobic exercise in never-treated hypertensive men.

KEY POINTS: Recent evidence indicates that metaboreflex regulates heart rate recovery after exercise (HRR). An increased metaboreflex activity during the post-exercise period might help to explain the reduced HRR observed in hypertensive subjects. Using lower limb circulatory occlusion, the present study showed that metaboreflex activation during the post-exercise period delayed HRR in never-treated hypertensive men compared to normotensives. These findings may be relevant for understanding the physiological mechanisms associated with autonomic dysfunction in hypertensive men. ABSTRACT: Muscle metaboreflex influences heart rate (HR) regulation after aerobic exercise. Therefore, increased metaboreflex sensitivity may help to explain the delayed HR recovery (HRR) reported in hypertension. The present study assessed and compared the effect of metaboreflex activation after exercise on HRR, cardiac baroreflex sensitivity (cBRS) and heart rate variability (HRV) in normotensive (NT) and hypertensive (HT) men. Twenty-three never-treated HT and 25 NT men randomly underwent two-cycle ergometer exercise sessions (30 min, 70% V̇O2 peak ) followed by 5 min of inactive recovery performed with (occlusion) or without (control) leg circulatory occlusion (bilateral thigh cuffs inflated to a suprasystolic pressure). HRR was assessed via HR reduction after 30, 60 and 300 s of recovery (HRR30s, HRR60s and HRR300s), as well as by the analysis of short- and long-term time constants of HRR. cBRS was assessed by sequence technique and HRV by the root mean square residual and the root mean square of successive differences between adjacent RR intervals on subsequent 30 s segments. Data were analysed using two- and three-way ANOVA. HRR60s and cBRS were significant and similarly reduced in both groups in the occlusion compared to the control session (combined values: 20 ± 10 vs. 26 ± 9 beats min-1 and 2.1 ± 1.2 vs. 3.2 ± 2.4 ms mmHg-1 , respectively, P < 0.05). HRR300s and HRV were also reduced in the occlusion session, although these reductions were significantly greater in HT compared to NT (-16 ± 11 vs. -8 ± 15 beats min-1 for HRR300s, P < 0.05). The results support the role of metaboreflex in HRR and suggest that increased metaboreflex sensitivity may partially explain the delayed HRR observed in HT men.

Prescrição de caminhada não supervisionada, risco cardiovascular e aptidão física / Unsupervised walking prescription, cardiovascular risk and physical fitness

OBJETIVO: Avaliar, numa situação real de atuação prática, o efeito da prescrição individualizada de caminhada sem supervisão da prática sobre o risco cardiovascular e a aptidão física de usuários de um parque público. MÉTODOS: 186 sujeitos (62 ± 10 anos) foram orientados a caminhar pelo menos 3x/sem, por 30 min, com intensidade de 50 a 80% da frequência cardíaca de reserva e a fazer alongamentos antes e após a caminhada. A aptidão física e os fatores de risco cardiovascular foram avaliados pré e pós-intervenção. A análise dos dados foi dividida em duas fases: 1) análise na amostra total; 2) análise nos indivíduos com fatores de risco alterados. Os dados foram comparados pelo teste t pareado. RESULTADOS: Na amostra total, a aptidão física melhorou nos testes de marcha estacionária (+8,1 ± 14,5 passos, p < 0,05), impulsão vertical (+0,5 ± 2,7 cm, p < 0,05), flexibilidade lombar (+1,1 ± 4,7 cm, p < 0,05) e flexibilidade de ombro (+1,2 ± 2,1 cm, p < 0,05). Não ocorreram alterações nos fatores de risco cardiovascular, com exceção da redução da pressão arterial diastólica (-0,9 ± 6,0 mmHg, p < 0,05). Entretanto, nos subgrupos com fatores alterados, observou-se reduções significantes das pressões arteriais sistólica e diastólica (-13,3 ± 16,9 e -5,8 ± 8,3 mmHg, p < 0,05, respectivamente) nos hipertensos, da colesterolemia total (-19,5 ± 33,5 mg/dl, p < 0,05) nos hipercolesterolêmicos e da circunferência da cintura (-1,0 ± 4,7 cm, p < 0,05) e do índice cintura-quadril (-0,01 ± 0,04, p < 0,05) nos com obesidade central. CONCLUSÃO: Numa situação real de atuação, a prescrição de caminhada sem supervisão da prática foi efetiva em melhorar a aptidão física da amostra geral e em diminuir o risco cardiovascular específico dos indivíduos com fatores de risco...

OBJECTIVE: To evaluate, at a real practical condition, the effects of individualized prescription of walking without supervision of practice on cardiovascular risk and fitness in users of a public park. METHODS: One hundred, eighty six subjects (62 ± 10 years) were instructed to walk at least 3 times/week, during 30min, at an intensity of 50-80% of heart rate reserve and encouraged to realize stretching exercises before and after walking. Physical fitness and cardiovascular risk factors were evaluated pre and post-intervention. Data analyze was divided in 2 phases: 1) role sample analysis; and 2) analysis on subjects with altered cardiovascular risk factors. Data were compared by paired t test. RESULTS: Considering the whole sample, physical fitness improved in the following tests: stationary gate (8.1 ± 14.5 paces, p < 0.05), vertical jump (0.5 ± 2.7 cm, p < 0.05), lumbar flexibility (1.1 ± 4.7 cm, p < 0.05) and shoulder flexibility (1.2 ± 2.1 cm, p < 0.05). No significant change was observed in cardiovascular risk factors, excepted by a reduction on diastolic blood pressure (-0.9 ± 6.0 mmHg, p < 0.05). On the other hand, considering the subjects with altered cardiovascular risk factors, a significant reduction was observed on systolic and diastolic blood pressures (-13.3 ± 16.9 and -5.8 ± 8.3 mmHg, p < 0.05, respectively) in hypertensive subjects, on total cholesterol (-19.5 ± 33.5 mg/dl, p < 0.05) in hypercholesterolemic subjects, and on waist circumference (-1.0 ± 4.7 cm, p < 0.05) and waist-hip index (0.01 ± 0.04, p < 0.05) in subjects with central obesity. CONCLUSION: Under real practical circumstances, the prescription of unsupervised walking was effective in improving physical fitness in general sample and in reducing the specific cardiovascular risk in subjects who have altered cardiovascular risk factors...